Literature DB >> 28597147

MiR-338-3p inhibits TNF-α-induced lipogenesis in human sebocytes.

Jia Liu1, Linggai Cao1, Yue Feng1, Yuhua Li1, Tao Li2.   

Abstract

OBJECTIVE: To suppress TNF-α-induced lipogenesis in sebocytes (associated with acne development) with microRNA-338-3p (miR-338-3p) and to explore the underlying mechanisms.
RESULTS: TNF-α increased lipid droplet formation in sebocytes which were used as in vitro model of inflammation-induced acne. Flow cytometry and TLC assays validated that miR-338-3p could suppress TNF-α-induced lipid droplet formation, down-regulate the expression of PREX2a, and inactivate AKT signaling in sebocytes. In addition, suppression of AKT activity by the PI3 K and AKT inhibitors diminished TNF-α-induced lipogenesis. PREX2a siRNA mimics the effects of miR-338-3p on AKT phosphorylation and lipogenesis. PREX2a overexpression consistently restored lipogenesis and AKT phosphorylation attenuated by miR-338-3p.
CONCLUSIONS: MiR-338-3p suppresses the TNF-α-induced lipogenesis in sebocytes by targeting PREX2a and down-regulating PI3K/AKT signaling.

Entities:  

Keywords:  Acne; Lipid droplets; Lipogenesis; MicroRNA-338-3p; PI3K/Akt; Sebocytes; TNF-α

Mesh:

Substances:

Year:  2017        PMID: 28597147     DOI: 10.1007/s10529-017-2369-3

Source DB:  PubMed          Journal:  Biotechnol Lett        ISSN: 0141-5492            Impact factor:   2.461


  6 in total

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5.  Circ_0038467 regulates lipopolysaccharide-induced inflammatory injury in human bronchial epithelial cells through sponging miR-338-3p.

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Review 6.  MicroRNA Cross-Involvement in Acne Vulgaris and Hidradenitis Suppurativa: A Literature Review.

Authors:  Francesco Borgia; Lucia Peterle; Paolo Custurone; Mario Vaccaro; Giovanni Pioggia; Sebastiano Gangemi
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  6 in total

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