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Literature DB >> 28533318

Putting the Brakes on Hypertensive Remodeling: An ATF3 Mechanism of Myofibroblast Restraint.

Abstract

Entities: Chemical Disease Gene Species

Keywords: Editorials; activating transcription factor 3; cardiac remodeling; fibrosis; hypertrophy; p38 mitogen-activated protein kinases

Mesh：

Substances：

Year: 2017 PMID： 28533318 PMCID： PMC6935562 DOI： 10.1161/CIRCULATIONAHA.117.028020

Source DB: PubMed Journal: Circulation ISSN： 0009-7322 Impact factor: 29.690

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12 in total

1. ATF3 expression in cardiomyocytes preserves homeostasis in the heart and controls peripheral glucose tolerance.

Authors: Roy Kalfon; Lilach Koren; Sharon Aviram; Ortal Schwartz; Tsonwin Hai; Ami Aronheim
Journal: Cardiovasc Res Date: 2016-11-07 Impact factor: 10.787

2. The regulation of ATF3 gene expression by mitogen-activated protein kinases.

Authors: Dan Lu; Jingchun Chen; Tsonwin Hai
Journal: Biochem J Date: 2007-01-15 Impact factor: 3.857

3. A TRPC6-dependent pathway for myofibroblast transdifferentiation and wound healing in vivo.

Authors: Jennifer Davis; Adam R Burr; Gregory F Davis; Lutz Birnbaumer; Jeffery D Molkentin
Journal: Dev Cell Date: 2012-09-27 Impact factor: 12.270

4. Transgenic mice with cardiac-specific expression of activating transcription factor 3, a stress-inducible gene, have conduction abnormalities and contractile dysfunction.

Authors: Y Okamoto; A Chaves; J Chen; R Kelley; K Jones; H G Weed; K L Gardner; L Gangi; M Yamaguchi; W Klomkleaw; T Nakayama; R L Hamlin; C Carnes; R Altschuld; J Bauer; T Hai
Journal: Am J Pathol Date: 2001-08 Impact factor: 4.307

Review 5. ATF3 transcription factor and its emerging roles in immunity and cancer.

Authors: Matthew R Thompson; Dakang Xu; Bryan R G Williams
Journal: J Mol Med (Berl) Date: 2009-08-25 Impact factor: 4.599

Review 6. Myofibroblasts: trust your heart and let fate decide.

Authors: Jennifer Davis; Jeffery D Molkentin
Journal: J Mol Cell Cardiol Date: 2013-11-02 Impact factor: 5.000

7. Activating transcription factor 3 deficiency promotes cardiac hypertrophy, dysfunction, and fibrosis induced by pressure overload.

Authors: Heng Zhou; Di-Fei Shen; Zhou-Yan Bian; Jing Zong; Wei Deng; Yan Zhang; Yuan-Yuan Guo; Hongliang Li; Qi-Zhu Tang
Journal: PLoS One Date: 2011-10-28 Impact factor: 3.240

Putting the Brakes on Hypertensive Remodeling: An ATF3 Mechanism of Myofibroblast Restraint.

1. ATF3 expression in cardiomyocytes preserves homeostasis in the heart and controls peripheral glucose tolerance.

2. The regulation of ATF3 gene expression by mitogen-activated protein kinases.

3. A TRPC6-dependent pathway for myofibroblast transdifferentiation and wound healing in vivo.

4. Transgenic mice with cardiac-specific expression of activating transcription factor 3, a stress-inducible gene, have conduction abnormalities and contractile dysfunction.

Review 5. ATF3 transcription factor and its emerging roles in immunity and cancer.

Review 6. Myofibroblasts: trust your heart and let fate decide.

7. Activating transcription factor 3 deficiency promotes cardiac hypertrophy, dysfunction, and fibrosis induced by pressure overload.

Review 8. Novel therapeutic strategies targeting fibroblasts and fibrosis in heart disease.

9. Adult cardiac expression of the activating transcription factor 3, ATF3, promotes ventricular hypertrophy.

10. MBNL1-mediated regulation of differentiation RNAs promotes myofibroblast transformation and the fibrotic response.

1. PDGF-D activation by macrophage-derived uPA promotes AngII-induced cardiac remodeling in obese mice.