| Literature DB >> 2841502 |
Abstract
Deletion analysis of the equine infectious anemia virus long terminal repeat revealed that sequences responsive to virus-specific transactivation are located within the region spanning the transcriptional start site (-31 to +22). In addition, an active exon of a trans-acting factor (tat) was identified downstream of pol and overlapping env (nucleotides 5264 to 5461). Activation by tat is accompanied by an increase in the steady-state levels of mRNA directed by the equine infectious anemia virus long terminal repeat.Entities:
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Year: 1988 PMID: 2841502 PMCID: PMC253482 DOI: 10.1128/JVI.62.9.3522-3526.1988
Source DB: PubMed Journal: J Virol ISSN: 0022-538X Impact factor: 5.103