Literature DB >> 28338586

Toward Microsurgical Correction of Cleft Lip Ex Utero through Restoration of Craniofacial Developmental Programs.

Xue Dong1,2, Wilmina N Landford1,2, James Hart1,2, Maurizio Risolino1,2, Omer Kaymakcalan1,2, Julia Jin1,2, Yoshiko Toyoda1,2, Elisabetta Ferretti1,2, Licia Selleri1,2, Jason A Spector1,2.   

Abstract

BACKGROUND: Cleft lip with or without cleft palate is present in approximately one in 500 to 700 live births, representing the most common congenital craniofacial anomaly. Previously, the authors developed a unique murine model with compound Pbx deficiency that exhibits fully penetrant cleft lip with or without cleft palate. To investigate the possibility of tissue repair at an early gestational stage, the authors designed a minimally invasive surgical approach suitable for intrauterine repair using Wnt9b-soaked collagen microspheres to restore craniofacial developmental programs for cleft correction.
METHODS: Collagen microspheres with diameters ranging from 20 to 50 μm were fabricated to serve as a delivery vehicle for Wnt9b. At gestational day 11.5, wild-type and Pbx-deficient murine embryos were isolated. Microspheres soaked in murine purified Wnt9b protein were microsurgically implanted at the midface lambdoidal junction. Embryos were cultured in a 37°C modified whole-embryo culture system.
RESULTS: Targeted release of Wnt9b resulted in augmented Wnt expression at the lambdoidal junction. Microsurgical implantation of Wnt9b-soaked microspheres resulted in cleft correction in 27.1 percent of the Pbx-deficient embryos. The difference in the ratio of the areas of clefting between implanted and nonimplanted embryos was significant (p < 0.05).
CONCLUSIONS: Ex utero correction of cleft lip with or without cleft palate in the authors' murine model by means of microsurgical intervention and targeted delivery of Wnt proteins is an innovative and promising strategy. Although further refinement and optimization of this technique will be required to improve efficacy, the authors believe that this approach will open new avenues toward unconventional prenatal interventions for patients with cleft lip with or without cleft palate, and provide future approaches for prenatal repair of other congenital head and neck disorders.

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Year:  2017        PMID: 28338586      PMCID: PMC5886781          DOI: 10.1097/PRS.0000000000003417

Source DB:  PubMed          Journal:  Plast Reconstr Surg        ISSN: 0032-1052            Impact factor:   4.730


  32 in total

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2.  Distinct functions for Bmp signaling in lip and palate fusion in mice.

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5.  Transgenes as probes for active chromosomal domains in mouse development.

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Review 6.  In utero surgery for cleft lip/palate: minimizing the "Ripple Effect" of scarring.

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7.  Hedgehog signaling in the neural crest cells regulates the patterning and growth of facial primordia.

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Review 9.  Fetal Cleft Lip/Palate Surgery: End of a Dream?

Authors:  Sinan Ozturk; Huseyin Karagoz; Fatih Zor; Gökhan Inangil; Kemal Kara
Journal:  Fetal Pediatr Pathol       Date:  2016-04-26       Impact factor: 0.958

10.  Requirement for Pbx1 in skeletal patterning and programming chondrocyte proliferation and differentiation.

Authors:  L Selleri; M J Depew; Y Jacobs; S K Chanda; K Y Tsang; K S Cheah; J L Rubenstein; S O'Gorman; M L Cleary
Journal:  Development       Date:  2001-09       Impact factor: 6.868

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1.  Identification of differentially expressed proteins involved in fetal scarless wound healing using a rat model of cleft lip.

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Journal:  Mol Med Rep       Date:  2021-06-24       Impact factor: 2.952

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