Literature DB >> 28327451

Hs-CRP and all-cause, cardiovascular, and cancer mortality risk: A meta-analysis.

Yunwei Li1, Xiaoming Zhong1, Guanchang Cheng2, Cuihua Zhao1, Lei Zhang1, Yan Hong1, Qilin Wan1, Ruili He1, Zhizhong Wang1.   

Abstract

BACKGROUND AND AIMS: Inconsistent findings have been reported on the association between high-sensitivity C-reactive protein (hs-CRP) and mortality risk. The objective of this meta-analysis was to investigate the association of elevated baseline hs-CRP levels with all-cause, cardiovascular, and cancer mortality risk in the general population.
METHODS: PubMed and Embase were systematically searched for studies published from inception to October 2016. Prospective observational studies were eligible if they reported the effects of elevated baseline hs-CRP levels on cancer-related, cardiovascular or all-cause mortality in the general population. The pooled adjusted risk ratio (RR) with 95% confidence interval (CI) comparing the highest to the lowest category of hs-CRP levels was used as association measures.
RESULTS: A total of 83,995 participants from 14 studies were identified. When comparing the highest to the lowest category of hs-CRP levels, the pooled RR was 1.25 (95% CI 1.13-1.38) for cancer-related mortality, 2.03 (95% CI 1.65-2.50) for cardiovascular mortality, and 1.75 (1.55-1.98) for all-cause mortality, respectively. Subgroup analysis showed that the effect of elevated hs-CRP levels on cancer-related mortality was observed in men (RR 1.26; 95% CI 1.11-1.43) but not in women (RR 1.03; 95% CI 0.83-1.27).
CONCLUSIONS: Elevated hs-CRP levels can independently predict risk of all-cause, cardiovascular mortality in the general population. However, the gender differences in the predictive role of hs-CRP on cancer mortality should to be further investigated.
Copyright © 2017 Elsevier B.V. All rights reserved.

Entities:  

Keywords:  All-cause mortality; Cardiovascular mortality; High-sensitivity C-reactive protein; Meta-analysis

Mesh:

Substances:

Year:  2017        PMID: 28327451     DOI: 10.1016/j.atherosclerosis.2017.02.003

Source DB:  PubMed          Journal:  Atherosclerosis        ISSN: 0021-9150            Impact factor:   5.162


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