Literature DB >> 28262193

Short-term Changes in Gal 3 Circulating Levels After Acute Myocardial Infarction.

Giulia Bivona1, Chiara Bellia1, Bruna Lo Sasso1, Luisa Agnello1, Concetta Scazzone1, Giuseppina Novo2, Marcello Ciaccio3.   

Abstract

BACKGROUND AND AIMS: Galectin 3 (Gal 3) is a β-galactoside-binding lectin known to play a part in inflammation, adverse remodeling and fibrosis. Gal 3 seems to be linked to atherogenesis and Coronary Artery Disease (CAD), but less is known about the relationship between Gal 3 and acute myocardial infarction (AMI). The aim of the present study is to assess circulating levels of Gal 3 after AMI and to evaluate short-term changes of the biomarker within 5 days from the acute event.
METHODS: Two hundred fifteen confirmed AMI patients (125 STEMI, M/F = 2.8; mean age: 65.4 ± 13.8 years) were enrolled in the present study; two blood samples were collected from each patient: first, within 1 h from admission to the Emergency Area (T1) and then upon discharge (T2).
RESULTS: Kinetics of Gal 3 during AMI show that the marker boosts during the acute event (T1) and then decreases from baseline, being significantly lower at T2 (18 [14.2-25] vs. 16.8 [12.7-23.4]; p = 0.006). Gal 3 levels were correlated to hsTnI and eGFR on admission (r = 0.2; p <0.001 and r = -0.25; p <0.001, respectively). Linear regression analysis confirms an association between Gal 3 and ejection fraction (r2 = 0.037; p = 0.005).
CONCLUSIONS: Gal 3 is reasonably supposed to be a part of those mechanisms leading to formation, destabilization and rupture of plaque; however, the usefulness of Gal 3 as a biomarker in CAD/AMI is far from being elucidated.
Copyright © 2016 IMSS. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  AMI; CAD; Galectin 3; Inflammation; Plaque

Mesh:

Substances:

Year:  2016        PMID: 28262193     DOI: 10.1016/j.arcmed.2016.12.009

Source DB:  PubMed          Journal:  Arch Med Res        ISSN: 0188-4409            Impact factor:   2.235


  12 in total

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