Literature DB >> 28245721

RAB7L1 promoter polymorphism and risk of Parkinson's disease; a case-control study.

Ali Khaligh1, Maryam Goudarzian1, Alireza Moslem2, Amirhosein Mehrtash3, Javad Jamshidi4, Hossein Darvish5, Babak Emamalizadeh6.   

Abstract

INTRODUCTION: Recent genome-wide association studies have explored some new loci in association with Parkinson's disease (PD). RAB7L1 is an important gene involved in one of the important neurological pathways, located in PARK16 locus. We performed a case-control study to examine the association between rs823144 SNP located in the promoter region of the RAB7L1 gene and PD risk in Iranian population.
METHODS: A total of 960 samples including 480 PD patients and 480 healthy controls were collected for analysis of the RAB7L1 rs823144 polymorphism using polymerase chain reaction-restriction fragment length polymorphism (PCR - RFLP) method.
RESULTS: We found significant differences in genotypic and allelic frequencies between patients and controls. Significant association was found between presence of minor allele (C) and decreased risk of PD development (p = 0.008, OR = 0.74 (0.605-0.924)). Also another significant association was observed between the CC genotype and PD (p = 0.004, OR = 0.441 (0.252-0.772)).
CONCLUSION: Our data support the association between rs823144 and decreased risk of PD.

Entities:  

Keywords:  Parkinson’s disease; RAB7L1; association; polymorphism; rs823144

Mesh:

Substances:

Year:  2017        PMID: 28245721     DOI: 10.1080/01616412.2017.1297558

Source DB:  PubMed          Journal:  Neurol Res        ISSN: 0161-6412            Impact factor:   2.448


  6 in total

Review 1.  Consequences of Rab GTPase dysfunction in genetic or acquired human diseases.

Authors:  Marcellus J Banworth; Guangpu Li
Journal:  Small GTPases       Date:  2017-12-28

2.  The atypical Rab GTPase associated with Parkinson's disease, Rab29, is localized to membranes.

Authors:  Yuki Nagai-Ito; Lejia Xu; Kyohei Ito; Yotaro Kajihara; Genta Ito; Taisuke Tomita
Journal:  J Biol Chem       Date:  2022-09-15       Impact factor: 5.486

3.  RAB7L1 Participates in Secondary Brain Injury Induced by Experimental Intracerebral Hemorrhage in Rats.

Authors:  Xiaoxing Tan; Yuchong Wei; Jie Cao; Degang Wu; Niansheng Lai; Ruming Deng; Haiying Li; Haitao Shen; Ya Peng; Xiang Li; Gang Chen
Journal:  J Mol Neurosci       Date:  2020-07-20       Impact factor: 3.444

4.  Methylation quantitative trait locus analysis of chronic postsurgical pain uncovers epigenetic mediators of genetic risk.

Authors:  Vidya Chidambaran; Xue Zhang; Valentina Pilipenko; Xiaoting Chen; Benjamin Wronowski; Kristie Geisler; Lisa J Martin; Artem Barski; Matthew T Weirauch; Hong Ji
Journal:  Epigenomics       Date:  2021-04-06       Impact factor: 4.778

Review 5.  Physiological and pathological functions of LRRK2: implications from substrate proteins.

Authors:  Miho Araki; Genta Ito; Taisuke Tomita
Journal:  Neuronal Signal       Date:  2018-10-10

6.  Distinct Roles for RAB10 and RAB29 in Pathogenic LRRK2-Mediated Endolysosomal Trafficking Alterations.

Authors:  Pilar Rivero-Ríos; Maria Romo-Lozano; Belén Fernández; Elena Fdez; Sabine Hilfiker
Journal:  Cells       Date:  2020-07-17       Impact factor: 6.600

  6 in total

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