Literature DB >> 28212877

Defining Outcomes for Clinical Trials of Leber Congenital Amaurosis Caused by GUCY2D Mutations.

Samuel G Jacobson1, Artur V Cideciyan2, Alexander Sumaroka2, Alejandro J Roman2, Jason Charng2, Monica Lu2, Shreyasi Choudhury3, Sharon B Schwartz2, Elise Heon4, Gerald A Fishman5, Shannon E Boye3.   

Abstract

PURPOSE: To determine outcome measures for a clinical trial of Leber congenital amaurosis (LCA) associated with mutations in the GUCY2D gene.
DESIGN: Retrospective observational case series.
METHODS: Twenty-eight patients with GUCY2D-LCA (aged 2-59 years) were studied clinically and with chromatic full-field sensitivity testing (FST), optical coherence tomography (OCT), pupillometry, and the NEI Visual Function Questionnaire (VFQ).
RESULTS: FST permitted quantitation of cone and rod sensitivity in these patients with severe visual impairment. For most patients, the degree of rod and cone sensitivity losses showed a relationship, thereby providing an opportunity to divide patients into cohorts by severity of rod and cone dysfunction. OCT analyses indicated that retinal structure could be used not only as an objective safety measure but also as an exploratory efficacy outcome. A foveal bulge was not present in 67% of patients. The intensity of inner segment/outer segment (ellipsoid zone line) reflectivity was reduced significantly at the fovea and in the rod-dense superior retina. Based on OCT and FST parameters, most patients had dissociation of structure and function. Abnormal pupillometry sensitivity in the majority of GUCY2D-LCA patients provided another objective efficacy outcome. NEI VFQ scores showed a similar range of findings to those of other severe retinal diseases.
CONCLUSION: Conventional outcome measures, such as visual acuity and the NEI VFQ, will need to be complemented by methods more specific to this GUCY2D-LCA population. Any therapeutic strategy should determine if there is an effect on rod as well as cone function and structure. FST provides a photoreceptor-based subjective outcome; and OCT in 2 retinal regions, fovea and superior retina, can assess photoreceptor structure. A change in the relationship of structure and function away from baseline becomes evidence of efficacy.
Copyright © 2017 Elsevier Inc. All rights reserved.

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Year:  2017        PMID: 28212877     DOI: 10.1016/j.ajo.2017.02.003

Source DB:  PubMed          Journal:  Am J Ophthalmol        ISSN: 0002-9394            Impact factor:   5.258


  12 in total

Review 1.  Leber's Congenital Amaurosis: Current Concepts of Genotype-Phenotype Correlations.

Authors:  Chu-Hsuan Huang; Chung-May Yang; Chang-Hao Yang; Yu-Chih Hou; Ta-Ching Chen
Journal:  Genes (Basel)       Date:  2021-08-19       Impact factor: 4.096

2.  Transient pupillary light reflex in CEP290- or NPHP5-associated Leber congenital amaurosis: Latency as a potential outcome measure of cone function.

Authors:  Arun K Krishnan; Samuel G Jacobson; Alejandro J Roman; Bhavya S Iyer; Alexandra V Garafalo; Elise Héon; Artur V Cideciyan
Journal:  Vision Res       Date:  2020-02-20       Impact factor: 1.886

3.  Pupillary Light Reflexes in Severe Photoreceptor Blindness Isolate the Melanopic Component of Intrinsically Photosensitive Retinal Ganglion Cells.

Authors:  Jason Charng; Samuel G Jacobson; Elise Heon; Alejandro J Roman; David B McGuigan; Rebecca Sheplock; Mychajlo S Kosyk; Malgorzata Swider; Artur V Cideciyan
Journal:  Invest Ophthalmol Vis Sci       Date:  2017-06-01       Impact factor: 4.799

Review 4.  Leber Congenital Amaurosis (LCA): Potential for Improvement of Vision.

Authors:  Artur V Cideciyan; Samuel G Jacobson
Journal:  Invest Ophthalmol Vis Sci       Date:  2019-04-01       Impact factor: 4.799

5.  GUCY2D-Associated Leber Congenital Amaurosis: A Retrospective Natural History Study in Preparation for Trials of Novel Therapies.

Authors:  Zaina Bouzia; Michalis Georgiou; Sarah Hull; Anthony G Robson; Kaoru Fujinami; Tryfon Rotsos; Nikolas Pontikos; Gavin Arno; Andrew R Webster; Alison J Hardcastle; Alessia Fiorentino; Michel Michaelides
Journal:  Am J Ophthalmol       Date:  2019-11-05       Impact factor: 5.258

6.  Treatment Potential for LCA5-Associated Leber Congenital Amaurosis.

Authors:  Katherine E Uyhazi; Puya Aravand; Brent A Bell; Zhangyong Wei; Lanfranco Leo; Leona W Serrano; Denise J Pearson; Ivan Shpylchak; Jennifer Pham; Vidyullatha Vasireddy; Jean Bennett; Tomas S Aleman
Journal:  Invest Ophthalmol Vis Sci       Date:  2020-05-11       Impact factor: 4.799

7.  The need for widely available genomic testing in rare eye diseases: an ERN-EYE position statement.

Authors:  Graeme C Black; Panagiotis Sergouniotis; Andrea Sodi; Bart P Leroy; Caroline Van Cauwenbergh; Petra Liskova; Karen Grønskov; Artur Klett; Susanne Kohl; Gita Taurina; Marius Sukys; Lonneke Haer-Wigman; Katarzyna Nowomiejska; João Pedro Marques; Dorothée Leroux; Frans P M Cremers; Elfride De Baere; Hélène Dollfus
Journal:  Orphanet J Rare Dis       Date:  2021-03-20       Impact factor: 4.123

8.  Leber Congenital Amaurosis Due to GUCY2D Mutations: Longitudinal Analysis of Retinal Structure and Visual Function.

Authors:  Samuel G Jacobson; Artur V Cideciyan; Alexander Sumaroka; Alejandro J Roman; Vivian Wu; Malgorzata Swider; Rebecca Sheplock; Arun K Krishnan; Alexandra V Garafalo
Journal:  Int J Mol Sci       Date:  2021-02-18       Impact factor: 5.923

9.  Safety and improved efficacy signals following gene therapy in childhood blindness caused by GUCY2D mutations.

Authors:  Samuel G Jacobson; Artur V Cideciyan; Allen C Ho; Igor V Peshenko; Alexandra V Garafalo; Alejandro J Roman; Alexander Sumaroka; Vivian Wu; Arun K Krishnan; Rebecca Sheplock; Sanford L Boye; Alexander M Dizhoor; Shannon E Boye
Journal:  iScience       Date:  2021-04-11

10.  Unexpected Genetic Cause in Two Female Siblings with High Myopia and Reduced Visual Acuity.

Authors:  M N Preising; C Friedburg; W Bowl; B Lorenz
Journal:  Biomed Res Int       Date:  2018-05-23       Impact factor: 3.411

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