Literature DB >> 28114842

Maternal plasma-soluble ST2 concentrations are elevated prior to the development of early and late onset preeclampsia - a longitudinal study.

Roberto Romero1,2,3,4, Piya Chaemsaithong1,5, Adi L Tarca1,5, Steven J Korzeniewski1,3,5, Eli Maymon1,5, Percy Pacora1,5, Bogdan Panaitescu1,5, Noppadol Chaiyasit1,5, Zhong Dong1,5, Offer Erez1,5, Sonia S Hassan1,5, Tinnakorn Chaiworapongsa1,5.   

Abstract

OBJECTIVE: The objectives of this study were to determine (1) the longitudinal profile of plasma soluble ST2 (sST2) concentrations in patients with preeclampsia and those with uncomplicated pregnancies; (2) whether the changes in sST2 occur prior to the diagnosis of preeclampsia; and (3) the longitudinal sST2 profile of women with early or late preeclampsia.
MATERIALS AND METHODS: This longitudinal nested case-control study included singleton pregnancies in the following groups: (1) uncomplicated pregnancies (n = 160); and (2) those complicated by early (<34 weeks, n = 9) and late (≥34 weeks, n = 31) preeclampsia. sST2 concentrations were determined by enzyme-linked immunosorbent assays. Mixed-effects models were used for the longitudinal analysis.
RESULTS: (1) Plasma sST2 concentration profiles across gestation differed significantly among cases and controls (p < 0.0001); (2) women with early preeclampsia had higher mean sST2 concentrations than controls at >22 weeks of gestation; cases with late preeclampsia had higher mean concentrations at >33 weeks of gestation (both p < 0.05); and (3) these changes started approximately 6 weeks prior to clinical diagnosis.
CONCLUSIONS: Maternal plasma sST2 concentrations are elevated 6 weeks prior to the clinical diagnosis of preeclampsia. An increase in the maternal plasma concentration of sST2 may contribute to an exaggerated intravascular inflammatory response and/or the Th1/Th2 imbalance in some cases.

Entities:  

Keywords:  Interleukin-1; Th1/Th2 immune response; interleukin-33; intravascular inflammation; prediction of preeclampsia

Mesh:

Substances:

Year:  2017        PMID: 28114842      PMCID: PMC5581264          DOI: 10.1080/14767058.2017.1286319

Source DB:  PubMed          Journal:  J Matern Fetal Neonatal Med        ISSN: 1476-4954


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