Literature DB >> 28100726

Decreased intestinal acetylcholinesterase in early Parkinson disease: An 11C-donepezil PET study.

Tatyana D Fedorova1, Louise B Seidelin2, Karoline Knudsen2, Anna C Schacht2, Jacob Geday2, Nicola Pavese2, David J Brooks2, Per Borghammer2.   

Abstract

OBJECTIVE: To investigate systemic levels of acetylcholinesterase in early Parkinson disease (PD) with 11C-donepezil PET, a potential marker of parasympathetic innervation.
METHODS: This was a cross-sectional study with 19 patients with early-stage PD (disease duration 1.5 ± 0.6 years) and 16 age-matched controls who had clinical assessments, olfaction tests, and 11C-donepezil PET to measure acetylcholinesterase density in peripheral organs.
RESULTS: The patients with PD showed significantly reduced 11C-donepezil uptake in the small intestine (-14%, p = 0.018), colon (-22%, p < 0.001), and kidneys (-14%, p = 0.028). No difference in myocardial or pancreatic acetylcholinesterase levels was seen.
CONCLUSION: We found significantly decreased 11C-donepezil signal in the intestine and kidneys of patients with early PD, suggesting that parasympathetic denervation is present early in the disease course.
© 2017 American Academy of Neurology.

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Year:  2017        PMID: 28100726     DOI: 10.1212/WNL.0000000000003633

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


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