Petter Bjornstad1, Richard J Johnson2, Janet K Snell-Bergeon3, Laura Pyle4, Asa Davis5, Nicole Foster6, David Z Cherney7, David M Maahs8. 1. Department of Pediatric Endocrinology, University of Colorado School of Medicine, Aurora, CO; Barbara Davis Center for Diabetes, University of Colorado Denver, Aurora, CO. Electronic address: Petter.Bjornstad@childrenscolorado.org. 2. Department of Nephrology, University of Colorado Denver, Aurora, CO. 3. Barbara Davis Center for Diabetes, University of Colorado Denver, Aurora, CO. 4. Department of Pediatric Endocrinology, University of Colorado School of Medicine, Aurora, CO. 5. Benaroya Research Institute, Seattle, WA. 6. Jaeb Center for Health Research Inc., Tampa, FL. 7. Department of Nephrology, University of Toronto School of Medicine, Toronto, ON. 8. Department of Pediatrics, Division of Endocrinology, Stanford University School of Medicine, Stanford, CA.
Abstract
BRIEF SUMMARY: Vasopressin exerts important cardio-renal effects, but remains problematic to measure. Copeptin is a more stable peptide derived from the same precursor molecule. In this case-control study from the Type 1 Diabetes Exchange (T1DX) Biobank registry, men with T1D and albuminuria had greater copeptin concentrations than men with normoalbuminuria.
BRIEF SUMMARY: Vasopressin exerts important cardio-renal effects, but remains problematic to measure. Copeptin is a more stable peptide derived from the same precursor molecule. In this case-control study from the Type 1 Diabetes Exchange (T1DX) Biobank registry, men with T1D and albuminuria had greater copeptin concentrations than men with normoalbuminuria.
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