Martyn J Matthews1, Daniel Green2, Helen Matthews2, Emma Swanwick2. 1. School of Health Sciences, University of Salford, Frederick Road, Salford, Greater Manchester, M6 6PU, UK. Electronic address: m.j.matthews@salford.ac.uk. 2. School of Health Sciences, University of Salford, Frederick Road, Salford, Greater Manchester, M6 6PU, UK.
Abstract
PURPOSE: To investigate the effects of training induced fatigue on shoulder strength, ROM, joint position sense, and stroke length in elite competitive swimmers. METHODS: Seventeen national level swimmers performed maximum isometric strength (internal and external rotation), ROM, and joint position sense tests before and after a fatiguing 8 × 100 m training set. Stroke length, heart rate, blood lactate and blood glucose levels were recorded throughout. RESULTS: Peak blood lactate, blood glucose levels, and heart rate increased significantly (P < 0.001) post-training confirming fatigue. Reductions were observed in stroke length of both arms (P < 0.001), external rotation range of motion (P < 0.001, -5.29°, Right shoulder; P = 0.04, -3.18°, Left shoulder) and joint position sense in their dominant (breathing side) arm (P = 0.03). CONCLUSIONS: This investigation revealed a reduction in stroke length across both arms and also an arm bias in swimming whereby a greater reduction in both external rotation range and joint position sense was observed in the dominant arm when fatigued. This has highlighted a relationship between fatigue and potential mechanism of shoulder pathology in swimmers.
PURPOSE: To investigate the effects of training induced fatigue on shoulder strength, ROM, joint position sense, and stroke length in elite competitive swimmers. METHODS: Seventeen national level swimmers performed maximum isometric strength (internal and external rotation), ROM, and joint position sense tests before and after a fatiguing 8 × 100 m training set. Stroke length, heart rate, blood lactate and blood glucose levels were recorded throughout. RESULTS: Peak blood lactate, blood glucose levels, and heart rate increased significantly (P < 0.001) post-training confirming fatigue. Reductions were observed in stroke length of both arms (P < 0.001), external rotation range of motion (P < 0.001, -5.29°, Right shoulder; P = 0.04, -3.18°, Left shoulder) and joint position sense in their dominant (breathing side) arm (P = 0.03). CONCLUSIONS: This investigation revealed a reduction in stroke length across both arms and also an arm bias in swimming whereby a greater reduction in both external rotation range and joint position sense was observed in the dominant arm when fatigued. This has highlighted a relationship between fatigue and potential mechanism of shoulder pathology in swimmers.
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