Literature DB >> 27505322

Effect of remote ischemic preconditioning on myocardial injury and inflammatory response induced by ablation for atrial fibrillation: A randomized controlled trial.

Ruijuan Han1, Xiaoqing Liu1, XianDong Yin1, Meili Zheng1, Kai Sun2, Xingpeng Liu1, Ying Tian1, Xinchun Yang3.   

Abstract

BACKGROUND: Remote ischemic preconditioning (RIPC) has been suggested to reduce postoperative release of cardiac and inflammatory markers in patients undergoing cardiac surgery. This study aimed to evaluate the effect of RIPC on nonischemic myocardial damage and inflammatory response in patients undergoing radiofrequency catheter ablation for paroxysmal atrial fibrillation (AF).
METHODS: Seventy-two patients with drug-refractory paroxysmal AF undergoing radiofrequency catheter ablation were randomized into RIPC or control groups. RIPC (intermittent arm ischemia through four cycles of 5-min inflation and 5-min deflation of a blood-pressure cuff) was performed once daily on 2 consecutive days before the ablation and immediately before ablation. Cardiac troponin-I (cTnI), high-sensitive C-reactive protein (hs-CRP), and interleukin (IL)-6 levels were measured before RIPC/sham RIPC, after the ablation, and 24 and 72h later. The early recurrence of atrial fibrillation (ERAF) in the two groups was observed over the subsequent 3months.
RESULTS: Radiofrequency ablation resulted in a significant rise in cTnI, hs-CRP, and IL-6 in both groups, which persisted for 72h. The RIPC group showed a lower increase in cTnI (P<0.001), hs-CRP (P=0.003), and IL-6 (P=0.008) than the control and tended to have a lower risk of ERAF (hazard ratio [HR]=0.77, 95% confidence interval [CI]: 0.32-1.88).
CONCLUSIONS: These results show that RIPC before ablation for paroxysmal AF significantly reduces the increase in cTnI, hs-CRP, and IL-6 associated with the procedure and results in a lower risk of ERAF. These findings suggest that RIPC could provide cardioprotection against nonischemic myocardial damage.
Copyright © 2016 Elsevier Ireland Ltd. All rights reserved.

Entities:  

Keywords:  Atrial fibrillation; Catheter ablation; Inflammatory response; Myocardial damage; Remote ischemic preconditioning

Mesh:

Substances:

Year:  2016        PMID: 27505322     DOI: 10.1016/j.ijcard.2016.07.229

Source DB:  PubMed          Journal:  Int J Cardiol        ISSN: 0167-5273            Impact factor:   4.164


  2 in total

1.  AKT and ERK1/2 activation via remote ischemic preconditioning prevents Kcne2-dependent sudden cardiac death.

Authors:  Zhaoyang Hu; Jin Liu; Leng Zhou; Xin Tian; Geoffrey W Abbott
Journal:  Physiol Rep       Date:  2019-02

Review 2.  Does remote ischaemic conditioning reduce inflammation? A focus on innate immunity and cytokine response.

Authors:  Lucie Pearce; Sean M Davidson; Derek M Yellon
Journal:  Basic Res Cardiol       Date:  2021-02-24       Impact factor: 17.165

  2 in total

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