Literature DB >> 27451003

Plasma thiols and thiol-disulfide homeostasis in patients with isolated coronary artery ectasia.

Emrullah Kızıltunç1, Murat Gök2, Harun Kundi2, Mustafa Çetin2, Canan Topçuoğlu2, Birsen Gülkan2, Hülya Çiçekçioğlu2, Ender Örnek2.   

Abstract

BACKGROUND AND AIMS: Thiol/disulfide homeostasis has an important role in the antioxidant defense system. Oxidative stress may contribute to the pathogenesis of coronary artery ectasia. The aim of this study was to evaluate plasma thiol levels and thiol/disulfide homeostasis in patients with isolated coronary artery ectasia.
METHODS: Forty-one patients with isolated coronary artery ectasia and 72 patients with normal coronary arteries were included in the study. Markis classification and number of ectatic coronary arteries were recorded. Plasma total thiol levels, native thiol levels and disulfide levels were measured. Thiol/disulfide homeostasis was appraised by calculating thiol/disulfide ratio.
RESULTS: Plasma native thiol levels were significantly lower (336.9 (252.9-374.1) vs. 353.1 (327.0-380.0), p = 0.041) and disulfide levels were significantly higher (18.9 ± 6.3 vs. 16.6 ± 3.4, p = 0.014) in patients with coronary artery ectasia than control patients. Both native thiol/disulfide and total thiol/disulfide ratio was significantly lower in the coronary artery ectasia group (p < 0.001). Multivariate logistic regression analysis revealed that native thiol levels, disulfide levels and native thiol/disulfide ratio were independently associated with the presence of coronary artery ectasia. Thiol/disulfide ratio was not different according to number of ectatic coronary arteries and there was no association between thiol/disulfide ratio and Markis classification.
CONCLUSIONS: Plasma thiol/disulfide homeostasis is altered in patients with coronary artery ectasia.
Copyright © 2016 Elsevier Ireland Ltd. All rights reserved.

Entities:  

Keywords:  Coronary artery ectasia; Oxidative stress; Thiol

Mesh:

Substances:

Year:  2016        PMID: 27451003     DOI: 10.1016/j.atherosclerosis.2016.07.904

Source DB:  PubMed          Journal:  Atherosclerosis        ISSN: 0021-9150            Impact factor:   5.162


  9 in total

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  9 in total

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