Literature DB >> 27445167

Renal Survival in Patients with Collapsing Compared with Not Otherwise Specified FSGS.

Louis-Philippe Laurin1,2, Adil M Gasim3, Vimal K Derebail1, JulieAnne G McGregor1,4, Jason M Kidd1,5, Susan L Hogan1, Caroline J Poulton1, Randal K Detwiler1, J Charles Jennette3, Ronald J Falk1, Patrick H Nachman6.   

Abstract

BACKGROUND AND OBJECTIVES: Idiopathic collapsing FSGS has historically been associated with poor renal outcomes. Minimal clinical data exist on the efficacy of immunosuppressive therapy. Our study sought to provide a comprehensive description of renal survival in patients with collapsing and not otherwise specified FSGS after controlling for factors affecting renal prognosis. DESIGN, SETTING, PARTICIPANTS, & MEASUREMENTS: We performed a retrospective analysis of an inception cohort study of patients diagnosed between 1989 and 2012. All potential patients with collapsing FSGS fulfilling the inclusion criteria were identified and compared with patients with not otherwise specified FSGS (approximately 1:2 ratio) on the basis of biopsy report and record availability. Time to ESRD was analyzed using Cox proportional hazards models.
RESULTS: In total, 187 patients were studied (61 collapsing and 126 not otherwise specified), with a mean follow-up of 96 months. At baseline, patients with collapsing FSGS had higher median proteinuria (12.2 [5.6-14.8] versus 4.4 [2.3-8.1] g/d, respectively; P<0.001), lower median albuminemia (2.4 [1.9-3.0] versus 2.9 [1.8-3.7] g/dl, respectively; P=0.12), and lower median eGFR (48 [26-73] versus 60 [42-92] ml/min per 1.73 m2, respectively; P=0.01) than patients with not otherwise specified FSGS. The proportion of patients with remission of proteinuria was similar in patients with collapsing FSGS and patients with not otherwise specified FSGS (65.7% [23 of 35] versus 63.2% [72 of 114], respectively; P=0.84). The overall renal outcome (ESRD defined as eGFR<15 ml/min per 1.73 m2, dialysis, or transplantation) of patients with collapsing FSGS was not poorer than that of patients with not otherwise specified FSGS in multivariate analyses after adjusting for baseline characteristics and immunotherapy (hazard ratio, 1.78; 95% confidence interval, 0.92 to 3.45).
CONCLUSIONS: Compared with not otherwise specified FSGS, idiopathic collapsing FSGS presented with more severe nephrotic syndrome and lower eGFR but had a similar renal survival after controlling for exposure to immunosuppressive treatment. These results highlight the importance of early diagnosis and institution of immunosuppressive therapy in patients with collapsing FSGS.
Copyright © 2016 by the American Society of Nephrology.

Entities:  

Keywords:  Follow-Up Studies; Humans; Kidney Failure, Chronic; Prognosis; Proportional Hazards Models; focal segmental glomerulosclerosis; glomerular filtration rate; glomerulonephritis; immunosuppression; nephrotic syndrome; proteinuria; renal dialysis

Mesh:

Substances:

Year:  2016        PMID: 27445167      PMCID: PMC5053801          DOI: 10.2215/CJN.13091215

Source DB:  PubMed          Journal:  Clin J Am Soc Nephrol        ISSN: 1555-9041            Impact factor:   8.237


  18 in total

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Authors:  Vivette D D'Agati; Frederick J Kaskel; Ronald J Falk
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Review 2.  Treatment of primary FSGS in adults.

Authors:  Stephen M Korbet
Journal:  J Am Soc Nephrol       Date:  2012-09-20       Impact factor: 10.121

3.  Histologic classification of FSGS: does form delineate function?

Authors:  Michael J Choi
Journal:  Clin J Am Soc Nephrol       Date:  2013-02-21       Impact factor: 8.237

4.  Changing etiologies of unexplained adult nephrotic syndrome: a comparison of renal biopsy findings from 1976-1979 and 1995-1997.

Authors:  M Haas; S M Meehan; T G Karrison; B H Spargo
Journal:  Am J Kidney Dis       Date:  1997-11       Impact factor: 8.860

5.  A more accurate method to estimate glomerular filtration rate from serum creatinine: a new prediction equation. Modification of Diet in Renal Disease Study Group.

Authors:  A S Levey; J P Bosch; J B Lewis; T Greene; N Rogers; D Roth
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6.  Association of histologic variants in FSGS clinical trial with presenting features and outcomes.

Authors:  Vivette D D'Agati; Joan M Alster; J Charles Jennette; David B Thomas; James Pullman; Daniel A Savino; Arthur H Cohen; Debbie S Gipson; Jennifer J Gassman; Milena K Radeva; Marva M Moxey-Mims; Aaron L Friedman; Frederick J Kaskel; Howard Trachtman; Charles E Alpers; Agnes B Fogo; Tom H Greene; Cynthia C Nast
Journal:  Clin J Am Soc Nephrol       Date:  2012-12-06       Impact factor: 8.237

7.  Nephrotic syndrome, progressive irreversible renal failure, and glomerular "collapse": a new clinicopathologic entity?

Authors:  M A Weiss; E Daquioag; E G Margolin; V E Pollak
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8.  Focal segmental glomerulosclerosis in nephrotic adults: presentation, prognosis, and response to therapy of the histologic variants.

Authors:  Micky J Chun; Stephen M Korbet; Melvin M Schwartz; Edmund J Lewis
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9.  Clinical and pathologic characteristics of focal segmental glomerulosclerosis pathologic variants.

Authors:  D B Thomas; N Franceschini; S L Hogan; S Ten Holder; C E Jennette; R J Falk; J C Jennette
Journal:  Kidney Int       Date:  2006-03       Impact factor: 10.612

10.  Collapsing glomerulopathy: a clinically and pathologically distinct variant of focal segmental glomerulosclerosis.

Authors:  R K Detwiler; R J Falk; S L Hogan; J C Jennette
Journal:  Kidney Int       Date:  1994-05       Impact factor: 10.612

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Journal:  J Am Soc Nephrol       Date:  2020-06-19       Impact factor: 10.121

2.  Focal Segmental Glomerulosclerosis, Risk Factors for End Stage Kidney Disease, and Response to Immunosuppression.

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3.  Validating a Computable Phenotype for Nephrotic Syndrome in Children and Adults Using PCORnet Data.

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4.  Clinical, Pathological, and Genetic Characteristics in Patients with Focal Segmental Glomerulosclerosis.

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Journal:  Kidney360       Date:  2022-05-24

5.  Digital spatial profiling of collapsing glomerulopathy.

Authors:  Kelly D Smith; David K Prince; Kammi J Henriksen; Roberto F Nicosia; Charles E Alpers; Shreeram Akilesh
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6.  Haemophagocytic lymphohistiocytosis with collapsing lupus podocytopathy as an unusual manifestation of systemic lupus erythematosus with APOL1 double-risk alleles.

Authors:  Bhavin Chokshi; Vivette D'Agati; Lilian Bizzocchi; Beverly Johnson; Barbara Mendez; Belinda Jim
Journal:  BMJ Case Rep       Date:  2019-01-14

7.  Collapsing focal segmental glomerulosclerosis in a patient with oral cavity cancer: A case report.

Authors:  Sae Byeol Choi; Kyoung Min Kim; Moon Hyang Park; Kyung Pyo Kang
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8.  COVID-19-Associated Glomerular Disease.

Authors:  Aneesha A Shetty; Ibrahim Tawhari; Luisa Safar-Boueri; Nay Seif; Ameen Alahmadi; Richard Gargiulo; Vikram Aggarwal; Irtaza Usman; Sergey Kisselev; Ali G Gharavi; Yahspal Kanwar; Susan E Quaggin
Journal:  J Am Soc Nephrol       Date:  2020-11-19       Impact factor: 14.978

9.  Severe Acute Respiratory Syndrome Coronavirus 2 Induced Focal Segmental Glomerulosclerosis.

Authors:  Is-Haq O Malik; Nida Ladiwala; Siddharth Chinta; Muhammad Khan; Komal Patel
Journal:  Cureus       Date:  2020-10-11

10.  A novel heterozygous variant of the COL4A4 gene in a Chinese family with hematuria and proteinuria leads to focal segmental glomerulosclerosis and chronic kidney disease.

Authors:  Liang-Liang Fan; Lv Liu; Fang-Mei Luo; Ran Du; Chen-Yu Wang; Yi Dong; Ji-Shi Liu
Journal:  Mol Genet Genomic Med       Date:  2020-11-07       Impact factor: 2.183

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