Literature DB >> 27402170

Prospective study on the potential of RAAS blockade to halt renal disease in Alport syndrome patients with heterozygous mutations.

Johanna Stock1, Johannes Kuenanz1, Niklas Glonke1, Joseph Sonntag1, Jenny Frese1, Burkhard Tönshoff2, Britta Höcker2, Bernd Hoppe3, Markus Feldkötter3, Lars Pape4, Christian Lerch4, Simone Wygoda5, Manfred Weber6, Gerhard-Anton Müller1, Oliver Gross7.   

Abstract

BACKGROUND: Patients with autosomal or X-linked Alport syndrome (AS) with heterozygous mutations in type IV collagen genes have a 1-20 % risk of progressing to end-stage renal disease during their lifetime. We evaluated the long-term renal outcome of patients at risk of progressive disease (chronic kidney disease stages 1-4) with/without nephroprotective therapy.
METHODS: This was a prospective, non-interventional, observational study which included data from a 4-year follow-up of AS patients with heterozygous mutations whose datasets had been included in an analysis of the 2010 database of the European Alport Registry. Using Kaplan-Meier estimates and logrank tests, we prospectively analyzed the updated datasets of 52 of these patients and 13 new datasets (patients added to the Registry after 2011). The effects of therapy, extrarenal symptoms and inheritance pattern on renal outcome were analyzed.
RESULTS: The mean prospective follow-up was 46 ± 10 months, and the mean time on therapy was 8.4 ± 4.4 (median 7; range 2-18) years. The time from the appearance of the first symptom to diagnosis was 8.1 ± 14.2 (range 0-52) years. At the time of starting therapy, 5.4 % of patients had an estimated glomerular filtration rate of <60 ml/min, 67.6 % had proteinuria and 27.0 % had microalbuminuria. Therapeutic strategies included angiotensin-converting enzymer inhibitors (97.1 %), angiotensin receptor antagonists (1 patient), dual therapy (11.8 %) and statins (8.8 %). Among patients included in the prospective dataset, prevented the need for dialysis. Among new patients, no patient at risk for renal failure progressed to the next disease stage after 4 years follow-up; three patients even regressed to a lower stage during therapy.
CONCLUSIONS: Treatment with blockers of the renin-angiotensin-aldosterone system prevents progressive renal failure in AS patients with heterozygous mutations in the genes causing AS. Considerable numbers of aging AS patients on dialysis may have heterozygous mutations in these genes (present in 1 % of total population) as underlying disease. Hence, greater alertness towards timely diagnosis and therapy has the potential to prevent progressive renal failure in most-if not all-AS patients with heterozygous mutations in the causal genes.

Entities:  

Keywords:  Alport syndrome; Chronic kidney disease; Collagen; Familial benign hematuria; Fibrosis; Renal insufficiency; Thin basement membrane disease

Mesh:

Year:  2016        PMID: 27402170     DOI: 10.1007/s00467-016-3452-z

Source DB:  PubMed          Journal:  Pediatr Nephrol        ISSN: 0931-041X            Impact factor:   3.714


  29 in total

1.  Genotype-phenotype correlation in X-linked Alport syndrome.

Authors:  Mir Reza Bekheirnia; Berenice Reed; Martin C Gregory; Kim McFann; Alireza Abdollah Shamshirsaz; Amirali Masoumi; Robert W Schrier
Journal:  J Am Soc Nephrol       Date:  2010-04-08       Impact factor: 10.121

Review 2.  Alport syndrome and the X chromosome: implications of a diagnosis of Alport syndrome in females.

Authors:  Clifford E Kashtan
Journal:  Nephrol Dial Transplant       Date:  2007-03-29       Impact factor: 5.992

3.  Thin-basement-membrane nephropathy in adults with persistent hematuria.

Authors:  A T Tiebosch; P M Frederik; P J van Breda Vriesman; J M Mooy; H van Rie; T W van de Wiel; J Wolters; E Zeppenfeldt
Journal:  N Engl J Med       Date:  1989-01-05       Impact factor: 91.245

Review 4.  The clinical features of thin basement membrane nephropathy.

Authors:  Martin C Gregory
Journal:  Semin Nephrol       Date:  2005-05       Impact factor: 5.299

5.  Incidence of renal failure and nephroprotection by RAAS inhibition in heterozygous carriers of X-chromosomal and autosomal recessive Alport mutations.

Authors:  Johanna Temme; Frederick Peters; Katharina Lange; Yves Pirson; Laurence Heidet; Roser Torra; Jean-Pierre Grunfeld; Manfred Weber; Christoph Licht; Gerhard-Anton Müller; Oliver Gross
Journal:  Kidney Int       Date:  2012-01-11       Impact factor: 10.612

6.  Meta-analysis of genotype-phenotype correlation in X-linked Alport syndrome: impact on clinical counselling.

Authors:  Oliver Gross; Kai-Olaf Netzer; Romy Lambrecht; Stefan Seibold; Manfred Weber
Journal:  Nephrol Dial Transplant       Date:  2002-07       Impact factor: 5.992

7.  Novel COL4A4 splice defect and in-frame deletion in a large consanguine family as a genetic link between benign familial haematuria and autosomal Alport syndrome.

Authors:  Oliver Gross; Kai-Olaf Netzer; Romy Lambrecht; Stefan Seibold; Manfred Weber
Journal:  Nephrol Dial Transplant       Date:  2003-06       Impact factor: 5.992

Review 8.  Alport syndrome from bench to bedside: the potential of current treatment beyond RAAS blockade and the horizon of future therapies.

Authors:  Oliver Gross; Laura Perin; Constantinos Deltas
Journal:  Nephrol Dial Transplant       Date:  2014-09       Impact factor: 5.992

9.  Clinical practice recommendations for the treatment of Alport syndrome: a statement of the Alport Syndrome Research Collaborative.

Authors:  Clifford E Kashtan; Jie Ding; Martin Gregory; Oliver Gross; Laurence Heidet; Bertrand Knebelmann; Michelle Rheault; Christoph Licht
Journal:  Pediatr Nephrol       Date:  2012-03-30       Impact factor: 3.714

10.  Frequency of COL4A3/COL4A4 mutations amongst families segregating glomerular microscopic hematuria and evidence for activation of the unfolded protein response. Focal and segmental glomerulosclerosis is a frequent development during ageing.

Authors:  Louiza Papazachariou; Panayiota Demosthenous; Myrtani Pieri; Gregory Papagregoriou; Isavella Savva; Christoforos Stavrou; Michael Zavros; Yiannis Athanasiou; Kyriakos Ioannou; Charalambos Patsias; Alexia Panagides; Costas Potamitis; Kyproula Demetriou; Marios Prikis; Michael Hadjigavriel; Maria Kkolou; Panayiota Loukaidou; Androulla Pastelli; Aristos Michael; Akis Lazarou; Maria Arsali; Loukas Damianou; Ioanna Goutziamani; Andreas Soloukides; Lakis Yioukas; Avraam Elia; Ioanna Zouvani; Polycarpos Polycarpou; Alkis Pierides; Konstantinos Voskarides; Constantinos Deltas
Journal:  PLoS One       Date:  2014-12-16       Impact factor: 3.240

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  4 in total

1.  Long-term treatment with EGFR inhibitor erlotinib attenuates renal inflammatory cytokines but not nephropathy in Alport syndrome mouse model.

Authors:  Kohei Omachi; Rui Miyakita; Ryosuke Fukuda; Yukari Kai; Mary Ann Suico; Tsubasa Yokota; Misato Kamura; Tsuyoshi Shuto; Hirofumi Kai
Journal:  Clin Exp Nephrol       Date:  2017-02-08       Impact factor: 2.801

2.  Diagnostic utility of genetic testing in patients undergoing renal biopsy.

Authors:  Katherine A Benson; Susan L Murray; Ross Doyle; Brendan Doyle; Anthony M Dorman; Denise Sadlier; Eoin Brennan; Margaret Large; Gianpiero L Cavalleri; Catherine Godson; Peter J Conlon
Journal:  Cold Spring Harb Mol Case Stud       Date:  2020-10-07

Review 3.  A practical approach to the genomics of kidney disorders.

Authors:  Eleanor Hay; Thomas Cullup; Angela Barnicoat
Journal:  Pediatr Nephrol       Date:  2021-03-06       Impact factor: 3.714

4.  Keratoconus in a patient with Alport syndrome: A case report.

Authors:  Majid Moshirfar; David F Skanchy; Aaron T Gomez; Yasmyne C Ronquillo; Benjamin Buckner; Phillip C Hoopes
Journal:  World J Clin Cases       Date:  2019-10-06       Impact factor: 1.337

  4 in total

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