Literature DB >> 27371487

Social disinhibition is a heritable subphenotype of tics in Tourette syndrome.

Matthew E Hirschtritt1, Sabrina M Darrow1, Cornelia Illmann1, Lisa Osiecki1, Marco Grados1, Paul Sandor1, Yves Dion1, Robert A King1, David L Pauls1, Cathy L Budman1, Danielle C Cath1, Erica Greenberg1, Gholson J Lyon1, Dongmei Yu1, Lauren M McGrath1, William M McMahon1, Paul C Lee1, Kevin L Delucchi1, Jeremiah M Scharf1, Carol A Mathews.   

Abstract

OBJECTIVE: To identify heritable symptom-based subtypes of Tourette syndrome (TS).
METHODS: Forty-nine motor and phonic tics were examined in 3,494 individuals (1,191 TS probands and 2,303 first-degree relatives). Item-level exploratory factor and latent class analyses (LCA) were used to identify tic-based subtypes. Heritabilities of the subtypes were estimated, and associations with clinical characteristics were examined.
RESULTS: A 6-factor exploratory factor analysis model provided the best fit, which paralleled the somatotopic representation of the basal ganglia, distinguished simple from complex tics, and separated out socially disinhibited and compulsive tics. The 5-class LCA model best distinguished among the following groups: unaffected, simple tics, intermediate tics without social disinhibition, intermediate with social disinhibition, and high rates of all tic types. Across models, a phenotype characterized by high rates of social disinhibition emerged. This phenotype was associated with increased odds of comorbid psychiatric disorders, in particular, obsessive-compulsive disorder and attention-deficit/hyperactivity disorder, earlier age at TS onset, and increased tic severity. The heritability estimate for this phenotype based on the LCA was 0.53 (SE 0.08, p 1.7 × 10(-18)).
CONCLUSIONS: Expanding on previous modeling approaches, a series of TS-related phenotypes, including one characterized by high rates of social disinhibition, were identified. These phenotypes were highly heritable and may reflect underlying biological networks more accurately than traditional diagnoses, thus potentially aiding future genetic, imaging, and treatment studies.
© 2016 American Academy of Neurology.

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Year:  2016        PMID: 27371487      PMCID: PMC4970665          DOI: 10.1212/WNL.0000000000002910

Source DB:  PubMed          Journal:  Neurology        ISSN: 0028-3878            Impact factor:   9.910


  21 in total

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1.  Genetic and phenotypic overlap of specific obsessive-compulsive and attention-deficit/hyperactive subtypes with Tourette syndrome.

Authors:  M E Hirschtritt; S M Darrow; C Illmann; L Osiecki; M Grados; P Sandor; Y Dion; R A King; D Pauls; C L Budman; D C Cath; E Greenberg; G J Lyon; D Yu; L M McGrath; W M McMahon; P C Lee; K L Delucchi; J M Scharf; C A Mathews
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