Literature DB >> 27197530

Combined effects of smoking and HPV16 in oropharyngeal cancer.

Devasena Anantharaman1, David C Muller1, Pagona Lagiou2, Wolfgang Ahrens3, Ivana Holcátová4, Franco Merletti5, Kristina Kjærheim6, Jerry Polesel7, Lorenzo Simonato8, Cristina Canova8, Xavier Castellsague9, Tatiana V Macfarlane10, Ariana Znaor11, Peter Thomson12, Max Robinson12, David I Conway13, Claire M Healy14, Anne Tjønneland15, Ulla Westin16, Johanna Ekström17, Jenny Chang-Claude18, Rudolf Kaaks18, Kim Overvad19, Dagmar Drogan20, Göran Hallmans21, Göran Laurell22, H B Bueno-de-Mesquita23, Petra H Peeters24, Antonio Agudo25, Nerea Larrañaga26, Ruth C Travis27, Domenico Palli28, Aurelio Barricarte29, Antonia Trichopoulou30, Saitakis George31, Dimitrios Trichopoulos32, J Ramón Quirós33, Sara Grioni34, Carlotta Sacerdote35, Carmen Navarro36, María-José Sánchez37, Rosario Tumino38, Gianluca Severi39, Marie-Christine Boutron-Ruault40, Francoise Clavel-Chapelon40, Salvatore Panico41, Elisabete Weiderpass42, Eiliv Lund43, Inger T Gram43, Elio Riboli44, Michael Pawlita45, Tim Waterboer45, Aimée R Kreimer46, Mattias Johansson1, Paul Brennan47.   

Abstract

BACKGROUND: Although smoking and HPV infection are recognized as important risk factors for oropharyngeal cancer, how their joint exposure impacts on oropharyngeal cancer risk is unclear. Specifically, whether smoking confers any additional risk to HPV-positive oropharyngeal cancer is not understood.
METHODS: Using HPV serology as a marker of HPV-related cancer, we examined the interaction between smoking and HPV16 in 459 oropharyngeal (and 1445 oral cavity and laryngeal) cancer patients and 3024 control participants from two large European multi-centre studies. Odds ratios and credible intervals [CrI], adjusted for potential confounders, were estimated using Bayesian logistic regression.
RESULTS: Both smoking [odds ratio (OR [CrI]: 6.82 [4.52, 10.29]) and HPV seropositivity (OR [CrI]: 235.69 [99.95, 555.74]) were independently associated with oropharyngeal cancer. The joint association of smoking and HPV seropositivity was consistent with that expected on the additive scale (synergy index [CrI]: 1.32 [0.51, 3.45]), suggesting they act as independent risk factors for oropharyngeal cancer.
CONCLUSIONS: Smoking was consistently associated with increase in oropharyngeal cancer risk in models stratified by HPV16 seropositivity. In addition, we report that the prevalence of oropharyngeal cancer increases with smoking for both HPV16-positive and HPV16-negative persons. The impact of smoking on HPV16-positive oropharyngeal cancer highlights the continued need for smoking cessation programmes for primary prevention of head and neck cancer.
© The Author 2016; all rights reserved. Published by Oxford University Press on behalf of the International Epidemiological Association.

Entities:  

Keywords:  Human papillomavirus; head and neck cancer risk; interaction; oropharynx cancer; tobacco smoking

Mesh:

Substances:

Year:  2016        PMID: 27197530      PMCID: PMC5841602          DOI: 10.1093/ije/dyw069

Source DB:  PubMed          Journal:  Int J Epidemiol        ISSN: 0300-5771            Impact factor:   7.196


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8.  Age, sexual behavior and human papillomavirus infection in oral cavity and oropharyngeal cancers.

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