Literature DB >> 27156057

Preclinical studies of VS-505: a non-absorbable highly effective phosphate binder.

J Ruth Wu-Wong1, Yung-Wu Chen1, Jonathan T Wong2, Jerry L Wessale1.   

Abstract

BACKGROUND AND
PURPOSE: Phosphate imbalance is often present in chronic kidney disease (CKD), and it contributes to a higher cardiovascular mortality rate. A phosphate binder is typically part of a treatment strategy for controlling phosphate imbalance. However, safety concerns and low compliance are two well-recognized disadvantages of on-market phosphate binders. This report describes the preclinical studies of VS-505, a non-absorbable, calcium- and aluminum-free, plant-derived polymer currently being evaluated in haemodialysis patients in Australia. EXPERIMENTAL APPROACH: Normal Sprague Dawley (SD) rats or uraemic SD rats induced by 5/6 nephrectomy fed a high-phosphate diet were treated with VS-505 or sevelamer (0.05-10% in food) for 5 and 28 days respectively. KEY
RESULTS: Urinary and serum phosphate levels were significantly elevated in untreated rats, and were decreased by VS-505 and sevelamer. VS-505 increased faecal phosphate levels in a dose-dependent manner. High-phosphate diet also caused an increase in serum FGF-23 and parathyroid hormone in nephrectomized (NX) rats, effects prevented by VS-505 or sevelamer. Significant aortic calcification was observed in NX rats treated with 5% sevelamer, whereas VS-505 at all doses tested did not show effects. VS-505 had no effects on small intestine histomorphology and intestinal sodium-dependent phosphate cotransporter gene expression. In vitro characterizations showed that VS-505 has a relatively high density and low expansion volume when exposed to simulated gastric fluid. CONCLUSIONS AND IMPLICATIONS: VS-505 is a safe and effective phosphate binder and may offer the advantage of having a reduced pill burden and minimal GI side effects for CKD patients.
© 2016 The British Pharmacological Society.

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Year:  2016        PMID: 27156057      PMCID: PMC4919576          DOI: 10.1111/bph.13510

Source DB:  PubMed          Journal:  Br J Pharmacol        ISSN: 0007-1188            Impact factor:   8.739


  51 in total

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3.  Effect of RenaGel, a non-absorbable, cross-linked, polymeric phosphate binder, on urinary phosphorus excretion in rats.

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4.  Serum phosphate and mortality in patients with chronic kidney disease.

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5.  Two novel vitamin D receptor modulators with similar structures exhibit different hypercalcemic effects in 5/6 nephrectomized uremic rats.

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Review 6.  Phosphate control in end-stage renal disease: barriers and opportunities.

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7.  Chronic kidney disease progression and outcome according to serum phosphorus in mild-to-moderate kidney dysfunction.

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8.  Differences in gastrointestinal calcium absorption after the ingestion of calcium-free phosphate binders.

Authors:  Geert J Behets; Geert Dams; Stephen J Damment; Patrick Martin; Marc E De Broe; Patrick C D'Haese
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9.  PA21, a new iron-based noncalcium phosphate binder, prevents vascular calcification in chronic renal failure rats.

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10.  Mapping the time-dependent effects of paricalcitol on serum calcium, phosphorus and parathyroid hormone levels in 5/6 nephrectomized uremic rats.

Authors:  J Ruth Wu-Wong; Masaki Nakane; Yung-wu Chen
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  2 in total

1.  Preclinical studies of VS-505: a non-absorbable highly effective phosphate binder.

Authors:  J Ruth Wu-Wong; Yung-Wu Chen; Jonathan T Wong; Jerry L Wessale
Journal:  Br J Pharmacol       Date:  2016-06-12       Impact factor: 8.739

2.  Effects of pharmacological inhibition of the sodium-dependent phosphate cotransporter 2b (NPT2b) on intestinal phosphate absorption in mouse and rat models.

Authors:  Xiaojun Wang; Yanping Xu; Xiaohong Yu; Asim Dey; Hong Y Zhang; Charity M Zink; Derek Wodka; Gina Porter; William F Matter; Leah Porras; Charles A Reidy; Jeffrey A Peterson; Brian E Mattioni; Joseph V Haas; Mark C Kowala; John R Wetterau
Journal:  Pharmacol Res Perspect       Date:  2022-04
  2 in total

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