The cricothyroid muscle in voicing control.

Initiation and maintenance of vibrations of the vocal folds require suitable conditions of adduction, longitudinal tension, and transglottal airflow. Thus manipulation of adduction/abduction, stiffening/slackening, or degree of transglottal flow may, in principle, be used to determine the voicing status of a speech segment. This study explores the control of voicing and voicelessness in speech with particular reference to the role of changes in the longitudinal tension of the vocal folds, as indicated by cricothyroid (CT) muscle activity. Electromyographic recordings were made from the CT muscle in two speakers of American English and one speaker of Dutch. The linguistic material consisted of reiterant speech made up of CV syllables where the consonants were voiced and voiceless stops, fricatives, and affricates. Comparison of CT activity associated with the voiced and voiceless consonants indicated a higher level for the voiceless consonants than for their voiced cognates. Measurements of the fundamental frequency (F0) at the beginning of a vowel following the consonant show the common pattern of higher F0 after voiceless consonants. For one subject, there was no difference in cricothyroid activity for voiced and voiceless affricates; in this case, the consonant-induced variations in the F0 of the following vowel were also less robust. Consideration of timing relationships between the EMG curves for voiced and voiceless consonants suggests that the differences most likely reflect control of vocal-fold tension for maintenance or suppression of phonatory vibrations. The same mechanism also seems to contribute to the well-known difference in F0 at the beginning of vowels following voiced and voiceless consonants.