Literature DB >> 26826201

Oncogenic PI3K mutations are as common as AKT1 and SMO mutations in meningioma.

Malak Abedalthagafi1, Wenya Linda Bi1, Ayal A Aizer1, Parker H Merrill1, Ryan Brewster1, Pankaj K Agarwalla1, Marc L Listewnik1, Dora Dias-Santagata1, Aaron R Thorner1, Paul Van Hummelen1, Priscilla K Brastianos1, David A Reardon1, Patrick Y Wen1, Ossama Al-Mefty1, Shakti H Ramkissoon1, Rebecca D Folkerth1, Keith L Ligon1, Azra H Ligon1, Brian M Alexander1, Ian F Dunn1, Rameen Beroukhim1, Sandro Santagata1.   

Abstract

BACKGROUND: Meningiomas are the most common primary intracranial tumor in adults. Identification of SMO and AKT1 mutations in meningiomas has raised the possibility of targeted therapies for some patients. The frequency of such mutations in clinical cohorts and the presence of other actionable mutations in meningiomas are important to define.
METHODS: We used high-resolution array-comparative genomic hybridization to prospectively characterize copy-number changes in 150 meningiomas and then characterized these samples for mutations in AKT1, KLF4, NF2, PIK3CA, SMO, and TRAF7.
RESULTS: Similar to prior reports, we identified AKT1 and SMO mutations in a subset of non-NF2-mutant meningiomas (ie, ∼9% and ∼6%, respectively). Notably, we detected oncogenic mutations in PIK3CA in ∼7% of non-NF2-mutant meningiomas. AKT1, SMO, and PIK3CA mutations were mutually exclusive. AKT1, KLF4, and PIK3CA mutations often co-occurred with mutations in TRAF7. PIK3CA-mutant meningiomas showed limited chromosomal instability and were enriched in the skull base.
CONCLUSION: This work identifies PI3K signaling as an important target for precision medicine trials in meningioma patients.
© The Author(s) 2016. Published by Oxford University Press on behalf of the Society for Neuro-Oncology. All rights reserved. For permissions, please e-mail: journals.permissions@oup.com.

Entities:  

Keywords:  AKT1; NF2; PIK3CA; SMO; aCGH; meningioma; molecular pathology

Mesh:

Substances:

Year:  2016        PMID: 26826201      PMCID: PMC4827048          DOI: 10.1093/neuonc/nov316

Source DB:  PubMed          Journal:  Neuro Oncol        ISSN: 1522-8517            Impact factor:   12.300


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7.  SWI/SNF chromatin remodeling complex alterations in meningioma.

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8.  DNA methylation analysis for the treatment of meningiomas.

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