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Literature DB >> 26817920

Alzheimer disease is in part a thrombohemorrhagic disorder.

A H Schmaier¹.

Abstract

Entities: Disease

Mesh：

Year: 2016 PMID： 26817920 DOI： 10.1111/jth.13277

Source DB: PubMed Journal: J Thromb Haemost ISSN： 1538-7836 Impact factor: 5.824

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Cited

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10 in total

1. Platelets are responsible for the accumulation of β-amyloid in blood clots inside and around blood vessels in mouse brain after thrombosis.

Authors: Lilia Y Kucheryavykh; Josué Dávila-Rodríguez; David E Rivera-Aponte; Lidia V Zueva; A Valance Washington; Priscilla Sanabria; Mikhail Y Inyushin
Journal: Brain Res Bull Date: 2016-11-28 Impact factor: 4.077

2. Aminopyrimidine Class Aggregation Inhibitor Effectively Blocks Aβ-Fibrinogen Interaction and Aβ-Induced Contact System Activation.

Authors: Pradeep K Singh; Masanori Kawasaki; Hanna E Berk-Rauch; Goushi Nishida; Takeshi Yamasaki; Michael A Foley; Erin H Norris; Sidney Strickland; Kazuyoshi Aso; Hyung Jin Ahn
Journal: Biochemistry Date: 2018-02-09 Impact factor: 3.162

Review 3. Amyloid β and free heme: bloody new insights into the pathogenesis of Alzheimer's disease.

Authors: Jörg Flemmig; Marcel Zámocký; A Alia
Journal: Neural Regen Res Date: 2018-07 Impact factor: 5.135

Review 4. Platelets in Neurodegenerative Conditions-Friend or Foe?

Authors: Odette Leiter; Tara L Walker
Journal: Front Immunol Date: 2020-05-05 Impact factor: 7.561

5. Accumulation of Amyloid Beta (Aβ) Peptide on Blood Vessel Walls in the Damaged Brain after Transient Middle Cerebral Artery Occlusion.

Authors: Antonio Henrique Martins; Astrid Zayas-Santiago; Yancy Ferrer-Acosta; Solianne M Martinez-Jimenez; Lidia Zueva; Amanda Diaz-Garcia; Mikhail Inyushin
Journal: Biomolecules Date: 2019-08-08

6. A network-informed analysis of SARS-CoV-2 and hemophagocytic lymphohistiocytosis genes' interactions points to Neutrophil extracellular traps as mediators of thrombosis in COVID-19.

Authors: Jun Ding; David Earl Hostallero; Mohamed Reda El Khili; Gregory Joseph Fonseca; Simon Milette; Nuzha Noorah; Myriam Guay-Belzile; Jonathan Spicer; Noriko Daneshtalab; Martin Sirois; Karine Tremblay; Amin Emad; Simon Rousseau
Journal: PLoS Comput Biol Date: 2021-03-08 Impact factor: 4.475

Alzheimer disease is in part a thrombohemorrhagic disorder.

1. Platelets are responsible for the accumulation of β-amyloid in blood clots inside and around blood vessels in mouse brain after thrombosis.

2. Aminopyrimidine Class Aggregation Inhibitor Effectively Blocks Aβ-Fibrinogen Interaction and Aβ-Induced Contact System Activation.

Review 3. Amyloid β and free heme: bloody new insights into the pathogenesis of Alzheimer's disease.

Review 4. Platelets in Neurodegenerative Conditions-Friend or Foe?

5. Accumulation of Amyloid Beta (Aβ) Peptide on Blood Vessel Walls in the Damaged Brain after Transient Middle Cerebral Artery Occlusion.

6. A network-informed analysis of SARS-CoV-2 and hemophagocytic lymphohistiocytosis genes' interactions points to Neutrophil extracellular traps as mediators of thrombosis in COVID-19.

Review 7. Beyond Haemostasis and Thrombosis: Platelets in Depression and Its Co-Morbidities.

Review 8. Aβ Peptide Originated from Platelets Promises New Strategy in Anti-Alzheimer's Drug Development.

9. Amyloid Beta Peptide Is Released during Thrombosis in the Skin.

Review 10. On the Role of Platelet-Generated Amyloid Beta Peptides in Certain Amyloidosis Health Complications.