Literature DB >> 26743751

En face OCT in Stargardt disease.

Andrea Sodi1, Dario Pasquale Mucciolo2, Francesca Cipollini1, Vittoria Murro1, Orsola Caporossi1, Gianni Virgili1, Stanislao Rizzo1.   

Abstract

PURPOSE: To evaluate the structural features of the macular region by enface OCT imaging in patients with clinical diagnosis of Stargardt disease, confirmed by the detection of ABCA4 mutations.
METHODS: Thirty-two STGD patients were included in the study for a total of 64 eyes. All patients received a comprehensive ophthalmological examination, color fundus photography, fundus auto-fluorescence imaging and OCT. Five OCT scans were considered: ILM and RPE scans (both automatically obtained from the instrument), above-RPE slab, photoreceptor slab and sub-RPE slab (these last three manually obtained).
RESULTS: ILM scans showed evident radial folds on the retinal surface in 8/64 eyes (12.5 %). In 6 of the 7 patients, these vitreo-retinal interface abnormalities were unilateral. The photoreceptor slab showed some macular alterations ranging from dis-homogeneous, hypo-reflective abnormalities (7/64 eyes, 11 %) to a homogeneous, well-defined, roundish, hypo-reflective area (17/64 eyes, 27 %) in all the eyes. The sub-RPE slab showed a centrally evident, hyper-reflective abnormality in 58/64 eyes (90.6 %). Superimposing the sub-RPE slab over the images corresponding to the photoreceptor slab, the area of the photoreceptor atrophy sharply exceeded that of the RPE atrophy (44/46 eyes, 96 %).
CONCLUSION: Enface OCT proved to be a clinically useful tool for the management of STGD patients, illustrating in vivo the structural abnormalities of the different retinal layers.

Entities:  

Keywords:  Dystrophy; Maculopathy; OCT; Photoreceptors; Retina; Stargardt

Mesh:

Substances:

Year:  2016        PMID: 26743751     DOI: 10.1007/s00417-015-3254-1

Source DB:  PubMed          Journal:  Graefes Arch Clin Exp Ophthalmol        ISSN: 0721-832X            Impact factor:   3.117


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6.  Novel mutations in of the ABCR gene in Italian patients with Stargardt disease.

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8.  Mutations in ABCA4 result in accumulation of lipofuscin before slowing of the retinoid cycle: a reappraisal of the human disease sequence.

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Review 9.  Gene therapy for Stargardt disease associated with ABCA4 gene.

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Review 2.  Clinical spectrum, genetic complexity and therapeutic approaches for retinal disease caused by ABCA4 mutations.

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3.  A Comparison of En Face Optical Coherence Tomography and Fundus Autofluorescence in Stargardt Disease.

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4.  Cross-Sectional and Longitudinal Assessment of the Ellipsoid Zone in Childhood-Onset Stargardt Disease.

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Review 5.  The role of multimodal imaging and vision function testing in ABCA4-related retinopathies and their relevance to future therapeutic interventions.

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