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Literature DB >> 26679098

Febuxostat-induced acute liver injury.

Matt Bohm¹, Raj Vuppalanchi¹, Naga Chalasani¹.

Abstract

Entities: Chemical Disease Gene Species

Mesh：

Substances：

Year: 2016 PMID： 26679098 PMCID： PMC4764455 DOI： 10.1002/hep.28403

Source DB: PubMed Journal: Hepatology ISSN： 0270-9139 Impact factor: 17.425

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4 in total

1. Febuxostat compared with allopurinol in patients with hyperuricemia and gout.

Authors: Michael A Becker; H Ralph Schumacher; Robert L Wortmann; Patricia A MacDonald; Denise Eustace; William A Palo; Janet Streit; Nancy Joseph-Ridge
Journal: N Engl J Med Date: 2005-12-08 Impact factor: 91.245

Review 2. Febuxostat: a selective xanthine oxidase inhibitor for the treatment of hyperuricemia and gout.

Authors: Susan P Bruce
Journal: Ann Pharmacother Date: 2006-11-28 Impact factor: 3.154

3. Clinical efficacy and safety of successful longterm urate lowering with febuxostat or allopurinol in subjects with gout.

Authors: Michael A Becker; H Ralph Schumacher; Patricia A MacDonald; Eric Lloyd; Christopher Lademacher
Journal: J Rheumatol Date: 2009-03-13 Impact factor: 4.666

4. Drug-Induced Liver Injury Network (DILIN) prospective study: rationale, design and conduct.

Authors: Robert J Fontana; Paul B Watkins; Herbert L Bonkovsky; Naga Chalasani; Timothy Davern; Jose Serrano; James Rochon
Journal: Drug Saf Date: 2009 Impact factor: 5.606

4 in total

9 in total

1. ISMP Adverse Drug Reactions: Sildenafil-Induced Erythema Multiforme Acute Liver Injury Due to Febuxostat Intravenous Acetaminophen-Induced Acute Hepatotoxicity Acute Transient Myopia Induced by Zanamivir Lidocaine-Induced Hoigne Syndrome.

Authors: Michael A Mancano
Journal: Hosp Pharm Date: 2016-12

2. Polyvinylpyrrolidone-Modified Taxifolin Liposomes Promote Liver Repair by Modulating Autophagy to Inhibit Activation of the TLR4/NF-κB Signaling Pathway.

Authors: Qiteng Ding; Wencong Liu; Xinglong Liu; Chuanbo Ding; Yingchun Zhao; Ling Dong; Huiying Chen; Shuwen Sun; Yiwen Zhang; Jinping Zhang; Ming Wu
Journal: Front Bioeng Biotechnol Date: 2022-06-01

Review 3. Recent Advances in the Histopathology of Drug-Induced Liver Injury.

Authors: David E Kleiner
Journal: Surg Pathol Clin Date: 2018-06

4. Isoorientin exerts a urate-lowering effect through inhibition of xanthine oxidase and regulation of the TLR4-NLRP3 inflammasome signaling pathway.

Authors: Meng-Fei An; Ming-Yue Wang; Chang Shen; Ze-Rui Sun; Yun-Li Zhao; Xuan-Jun Wang; Jun Sheng
Journal: J Nat Med Date: 2020-11-13 Impact factor: 2.343

Febuxostat-induced acute liver injury.

1. Febuxostat compared with allopurinol in patients with hyperuricemia and gout.

Review 2. Febuxostat: a selective xanthine oxidase inhibitor for the treatment of hyperuricemia and gout.

3. Clinical efficacy and safety of successful longterm urate lowering with febuxostat or allopurinol in subjects with gout.

4. Drug-Induced Liver Injury Network (DILIN) prospective study: rationale, design and conduct.

1. ISMP Adverse Drug Reactions: Sildenafil-Induced Erythema Multiforme Acute Liver Injury Due to Febuxostat Intravenous Acetaminophen-Induced Acute Hepatotoxicity Acute Transient Myopia Induced by Zanamivir Lidocaine-Induced Hoigne Syndrome.

2. Polyvinylpyrrolidone-Modified Taxifolin Liposomes Promote Liver Repair by Modulating Autophagy to Inhibit Activation of the TLR4/NF-κB Signaling Pathway.

Review 3. Recent Advances in the Histopathology of Drug-Induced Liver Injury.

4. Isoorientin exerts a urate-lowering effect through inhibition of xanthine oxidase and regulation of the TLR4-NLRP3 inflammasome signaling pathway.

Review 5. Side Effects and Interactions of the Xanthine Oxidase Inhibitor Febuxostat.

Review 6. From purines to purinergic signalling: molecular functions and human diseases.

Review 7. Hyperuricemia-Related Diseases and Xanthine Oxidoreductase (XOR) Inhibitors: An Overview.

8. Concomitant febuxostat enhances methotrexate-induced hepatotoxicity by inhibiting breast cancer resistance protein.

9. N-Butyrylated hyaluronic acid ameliorates gout and hyperuricemia in animal models.