Stefan Renvert1,2,3, Marc Quirynen4. 1. Department of Oral Health Sciences, Kristianstad University, Kristianstad, Sweden. 2. School of Dental Sciences, Trinity College, Dublin, Ireland. 3. Blekinge Institute of Technology, Karlskrona, Sweden. 4. Department of Oral Health Sciences, Katholieke Universiteit Leuven, University Hospitals Leuven, Leuven, Belgium.
Abstract
AIM: To examine the existing evidence in identifying risk indicators in the etiology of peri-implantitis. MATERIAL AND METHODS: A literature search was performed in MEDLINE via PubMed database of the US National Library of Medicine, for articles published until October 2014 using Medical Subject Heading search terms + free text terms and in different combinations. RESULTS: The microbiota associated with peri-implantitis is complex, demonstrating differences and similarities to the one seen at periodontitis sites. Plaque accumulation at dental implants triggers the inflammatory response leading to peri-implant mucositis/peri-implantitis. Individuals with a history of periodontal disease and smokers have an increased risk of developing peri-implantitis. There is some evidence to support the role of genetic polymorphism, diabetes, and excess cement as risk indicators for the development of peri-implantitis. There is also evidence to support that individuals on regular maintenance are less likely to develop peri-implantitis and that successful treatment of periodontitis prior to implant placement lowers the risk of peri-implantitis. CONCLUSIONS: Plaque accumulation at implants will result in the development of an inflammation at implants. A history of periodontal disease, smoking, excess cement, and lack of supportive therapy should be considered as risk indicators for the development of peri-implantitis.
AIM: To examine the existing evidence in identifying risk indicators in the etiology of peri-implantitis. MATERIAL AND METHODS: A literature search was performed in MEDLINE via PubMed database of the US National Library of Medicine, for articles published until October 2014 using Medical Subject Heading search terms + free text terms and in different combinations. RESULTS: The microbiota associated with peri-implantitis is complex, demonstrating differences and similarities to the one seen at periodontitis sites. Plaque accumulation at dental implants triggers the inflammatory response leading to peri-implant mucositis/peri-implantitis. Individuals with a history of periodontal disease and smokers have an increased risk of developing peri-implantitis. There is some evidence to support the role of genetic polymorphism, diabetes, and excess cement as risk indicators for the development of peri-implantitis. There is also evidence to support that individuals on regular maintenance are less likely to develop peri-implantitis and that successful treatment of periodontitis prior to implant placement lowers the risk of peri-implantitis. CONCLUSIONS: Plaque accumulation at implants will result in the development of an inflammation at implants. A history of periodontal disease, smoking, excess cement, and lack of supportive therapy should be considered as risk indicators for the development of peri-implantitis.
Authors: Mariane B Sordi; Vittoria Perrotti; Flavia Iaculli; Keila C R Pereira; Ricardo S Magini; Stefan Renvert; Stefano Antonio Gattone; Adriano Piattelli; Marco A Bianchini Journal: Clin Oral Investig Date: 2020-11-05 Impact factor: 3.573