Literature DB >> 26313020

Evaluation of MicroRNA-146a and Its Targets in Gingival Tissues of Patients With Chronic Periodontitis.

Hossein Motedayyen1, Somayeh Ghotloo2, Mahmood Saffari3, Mandana Sattari1, Reza Amid4.   

Abstract

BACKGROUND: MicroRNAs (miRNAs) are a group of small non-coding RNAs that play an important role in the regulation of gene expression. miRNA-146a (miR-146a), a member of the miR-146 family, is involved in the control of inflammation. Periodontitis is a set of chronic inflammatory disorders of the tissues surrounding the teeth that lead to the breakdown of alveolar bone and tooth loss. In this study, expression levels of miR-146a and its targets, including tumor necrosis factor (TNF)-α, interleukin (IL)-1β, and IL-6, are evaluated in human patients with chronic periodontitis (CP).
METHODS: The study population consisted of 10 healthy controls and 20 individuals with CP. For each participant, clinical parameters including probing depth and clinical attachment level were measured, and a gingival tissue sample was collected. Levels of miR-146a, TNF-α, IL-1β, and IL-6 were quantified using real-time polymerase chain reaction.
RESULTS: Levels of miR-146a were significantly higher in patients with CP (P <0.001). There was a positive correlation between levels of miR-146a and clinical parameters (P <0.05). Elevated miR-146a was accompanied by a significant reduction in TNF-α and IL-6 (P <0.001).
CONCLUSIONS: Patients with CP had higher levels of miR-146a than healthy individuals, accompanied by reduced levels of TNF-α and IL-6. A positive relationship between miR-146a levels and clinical parameters suggests a pathophysiologic role of miR-146a in CP.

Entities:  

Keywords:  Chronic periodontitis; cytokines; interleukin-1beta; interleukin-6; micrornas; tumor necrosis factor-alpha

Mesh:

Substances:

Year:  2015        PMID: 26313020     DOI: 10.1902/jop.2015.150319

Source DB:  PubMed          Journal:  J Periodontol        ISSN: 0022-3492            Impact factor:   6.993


  28 in total

1.  Dysregulation of human miRNAs and increased prevalence of HHV miRNAs in obese periodontitis subjects.

Authors:  Afsar R Naqvi; Maria F Brambila; Gloria Martínez; Gabriela Chapa; Salvador Nares
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2.  microRNA-142 is upregulated by tumor necrosis factor-alpha and triggers apoptosis in human gingival epithelial cells by repressing BACH2 expression.

Authors:  Song Li; Zhongchen Song; Jiachen Dong; Rong Shu
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3.  Comparative evaluation of microRNA-155 expression level and its correlation with tumor necrotizing factor α and interleukin 6 in patients with chronic periodontitis.

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Review 4.  Emerging role of epigenetic regulations in periodontitis: a literature review.

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Journal:  Am J Transl Res       Date:  2022-04-15       Impact factor: 3.940

5.  Expression of miR-155 and miR-146a in the saliva of patients with periodontitis and its clinical value.

Authors:  Peng Wu; Jie Feng; Wei Wang
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Authors:  Liu Hong; Hongli Sun; Brad A Amendt
Journal:  Bone       Date:  2020-12-09       Impact factor: 4.398

7.  MicroRNA expression profile of human periodontal ligament cells under the influence of Porphyromonas gingivalis LPS.

Authors:  Anqing Du; Sen Zhao; LingYun Wan; TianTao Liu; Zaoxia Peng; ZiYu Zhou; Zhengyu Liao; Huan Fang
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Review 8.  MicroRNAs: Harbingers and shapers of periodontal inflammation.

Authors:  Xianghong Luan; Xiaofeng Zhou; Pooria Fallah; Mirali Pandya; Huling Lyu; Deborah Foyle; Dan Burch; Thomas G H Diekwisch
Journal:  Semin Cell Dev Biol       Date:  2021-06-10       Impact factor: 7.499

Review 9.  MicroRNAs as Salivary Markers for Periodontal Diseases: A New Diagnostic Approach?

Authors:  Gerhard Schmalz; Simin Li; Ralph Burkhardt; Sven Rinke; Felix Krause; Rainer Haak; Dirk Ziebolz
Journal:  Biomed Res Int       Date:  2016-06-27       Impact factor: 3.411

10.  ET-1 Promotes Differentiation of Periodontal Ligament Stem Cells into Osteoblasts through ETR, MAPK, and Wnt/β-Catenin Signaling Pathways under Inflammatory Microenvironment.

Authors:  Li Liang; Wei Zhou; Nan Yang; Jifeng Yu; Hongchen Liu
Journal:  Mediators Inflamm       Date:  2016-01-13       Impact factor: 4.711

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