Literature DB >> 26226245

Elevated plasma and cerebrospinal fluid interleukin-1 beta and tumor necrosis factor-alpha concentration and combined outcome of death or abnormal neuroimaging in preterm neonates with early-onset clinical sepsis.

S Basu1, P Agarwal1, S Anupurba2, R Shukla3, A Kumar1.   

Abstract

OBJECTIVE: Prematurity and sepsis are the major contributors of neonatal mortality and neurodevelopmental sequelae. The present study was conducted to measure the plasma and cerebrospinal fluid (CSF) concentration of interleukin (IL)-1β and tumor necrotic factor (TNF)-α in preterm neonates with early-onset clinical sepsis (EOCS), and to find out their association with combined outcome of death or abnormal neuroimaging. STUDY
DESIGN: Thirty-two preterm (⩽34 weeks) neonates with EOCS and 32 gestational age-matched, healthy neonates served as cases and controls, respectively. Samples were collected soon after birth. Neonates were followed up clinically and by serial cranial ultrasonography (CUS) until discharge and subsequently by magnetic resonance imaging (MRI) of brain until 1 year. Developmental screening was done by Denver Developmental Screening test-II. RESULT: In EOCS group, no neonate had any clinical/microbiological evidence of meningitis. Blood culture was positive in 17 (53%). CUS was abnormal in 12 (37%) (intracranial hemorrhage-11, periventricular leukomalacia-1). Ten (31%) neonates expired. Significant elevation of plasma and CSF IL-1β and TNF-α was observed in the EOCS group. On follow-up, seven (22%) neonates showed evidence of white matter damage in MRI, two of them had developmental delay and microcephaly. Plasma and CSF IL-1β and TNF-α concentration were significantly elevated in deceased neonates and those with abnormal neuroimaging. Both biomarkers demonstrated high predictive accuracy for poor outcome in receiver-operating curve analysis.
CONCLUSION: Elevation of plasma and CSF IL-1β and TNF-α is associated with an increase in the combined outcome of death or abnormal neuroimaging in preterm neonates with EOCS in the absence of clinical/microbiological evidence of meningitis with high predictive accuracy.

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Year:  2015        PMID: 26226245     DOI: 10.1038/jp.2015.86

Source DB:  PubMed          Journal:  J Perinatol        ISSN: 0743-8346            Impact factor:   2.521


  46 in total

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3.  Correlation between placental histopathology and fetal/neonatal outcome: chorioamnionitis and funisitis are associated to intraventricular haemorrage and retinopathy of prematurity in preterm newborns.

Authors:  Francesca Moscuzza; Francesca Belcari; Vincenzo Nardini; Ambra Bartoli; Chiara Domenici; Armando Cuttano; Paolo Ghirri; Antonio Boldrini
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Review 4.  Neonatal infection and long-term neurodevelopmental outcome in the preterm infant.

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5.  Clinical chorioamnionitis and the prognosis for very low birth weight infants.

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Review 6.  Chorioamnionitis as a risk factor for cerebral palsy: A meta-analysis.

Authors:  Y W Wu; J M Colford
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7.  Inflammation-induced preterm birth alters neuronal morphology in the mouse fetal brain.

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8.  Preventing preterm births: analysis of trends and potential reductions with interventions in 39 countries with very high human development index.

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Review 2.  An Integrative Review of Cytokine/Chemokine Predictors of Neurodevelopment in Preterm Infants.

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3.  Dose-dependent structural and immunological changes in the placenta and fetal brain in response to systemic inflammation during pregnancy.

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4.  The significance of polymorphisms in genes encoding Il-1β, Il-6, TNFα, and Il-1RN in the pathogenesis of intraventricular hemorrhage in preterm infants.

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5.  Microglia-derived IL-1β contributes to axon development disorders and synaptic deficit through p38-MAPK signal pathway in septic neonatal rats.

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6.  Plasma Tumor Necrosis Factor-alpha (TNF-α) Levels Correlate with Disease Severity in Spastic Diplegia, Triplegia, and Quadriplegia in Children with Cerebral Palsy.

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8.  In Premature Newborns Intraventricular Hemorrhage Causes Cerebral Vasospasm and Associated Neurodisability via Heme-Induced Inflammasome-Mediated Interleukin-1 Production and Nitric Oxide Depletion.

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9.  Cerebrospinal fluid protein and glucose levels in neonates with a systemic inflammatory response without meningitis.

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10.  The Cerebrospinal Fluid Inflammatory Response to Preterm Birth.

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Journal:  Front Physiol       Date:  2018-09-12       Impact factor: 4.566

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