Literature DB >> 26218678

Fludarabine, cyclophosphamide, and multiple-dose rituximab as frontline therapy for chronic lymphocytic leukemia.

Nicholas J Short1, Michael J Keating2, William G Wierda2, Stefan Faderl3, Alessandra Ferrajoli2, Zeev Estrov2, Susan C Smith2, Susan M O'Brien4.   

Abstract

BACKGROUND: Fludarabine, cyclophosphamide, and rituximab (FCR) result in durable responses in patients with previously untreated chronic lymphocytic leukemia (CLL). Previous reports have suggested that in patients with relapsed CLL, a dose-intensified rituximab regimen increases response rates in comparison with standard-dose rituximab. It is unknown whether rituximab intensification of the FCR regimen will result in improved response rates and patient outcomes in patients with previously untreated CLL.
METHODS: A single-arm study was conducted to evaluate the safety and efficacy of a modified FCR regimen with multiple-dose rituximab (FCR3) in 65 patients with previously untreated CLL. The results were compared with those for a historical cohort treated with FCR.
RESULTS: The overall response rate to FCR3 was 97%, with 75% of the patients achieving a complete remission. Minimal residual disease negativity was achieved for 62% of the patients according to flow cytometry. The median time to progression (TTP) was 81 months, and the median overall survival (OS) was not reached, with 58% of the patients still alive at a median survivor follow-up of 9.7 years. Grade 3 or 4 neutropenia, grade 3 or 4 thrombocytopenia, and major infections were observed with 45%, 5%, and 1.9% of the FCR3 courses, respectively. Therapy-related myelodysplastic syndrome (t-MDS) or therapy-related acute myelogenous leukemia (t-AML) developed in 7 patients (11%; P < .01 vs the historical FCR cohort).
CONCLUSIONS: In patients with previously untreated CLL, FCR3 resulted in response rates, TTP, and OS similar to those of a historical cohort of patients treated with FCR. FCR3 was associated with an increased incidence of t-MDS/t-AML.
© 2015 American Cancer Society.

Entities:  

Keywords:  and rituximab (FCR); chemoimmunotherapy; chronic lymphocytic leukemia; cyclophosphamide; fludarabine; rituximab; therapy-related acute myelogenous leukemia; therapy-related myelodysplastic syndrome

Mesh:

Substances:

Year:  2015        PMID: 26218678      PMCID: PMC4824541          DOI: 10.1002/cncr.29605

Source DB:  PubMed          Journal:  Cancer        ISSN: 0008-543X            Impact factor:   6.860


  25 in total

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2.  Fludarabine, cyclophosphamide and rituximab plus granulocyte macrophage colony-stimulating factor as frontline treatment for patients with chronic lymphocytic leukemia.

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Authors:  Sebastian Böttcher; Matthias Ritgen; Kirsten Fischer; Stephan Stilgenbauer; Raymonde M Busch; Günter Fingerle-Rowson; Anna Maria Fink; Andreas Bühler; Thorsten Zenz; Michael Karl Wenger; Myriam Mendila; Clemens-Martin Wendtner; Barbara F Eichhorst; Hartmut Döhner; Michael J Hallek; Michael Kneba
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9.  Adoptive transfer of tumor reactive B cells confers host T-cell immunity and tumor regression.

Authors:  Qiao Li; Xiangming Lao; Qin Pan; Ning Ning; Ji Yet; Yingxin Xu; Shengping Li; Alfred E Chang
Journal:  Clin Cancer Res       Date:  2011-06-20       Impact factor: 12.531

10.  Potentiation of fludarabine cytotoxicity on non-Hodgkin's lymphoma by pentoxifylline and rituximab.

Authors:  S Alas; B Bonavida; C Emmanouilides
Journal:  Anticancer Res       Date:  2000 Sep-Oct       Impact factor: 2.480

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Review 5.  Trial watch: chemotherapy-induced immunogenic cell death in immuno-oncology.

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