Literature DB >> 26178600

Potential involvement of IL-9 and Th9 cells in the pathogenesis of rheumatoid arthritis.

Francesco Ciccia1, Giuliana Guggino2, Aroldo Rizzo3, Antonio Manzo4, Barbara Vitolo4, Marco Pio La Manna5, Giuseppina Giardina5, Guido Sireci5, Francesco Dieli5, Carlo Maurizio Montecucco4, Riccardo Alessandro5, Giovanni Triolo6.   

Abstract

OBJECTIVE: IL-9 has been shown to be upregulated before the clinical onset of articular disease in RA. The exact role of IL-9 and Th9 cells in RA, however, has not yet been adequately studied. The aim of this study was to evaluate the expression of IL-9 and IL-9-expressing cells in RA patients.
METHODS: IL-9, IL-9R, PU.1, IL-9, thymic stromal lymphopoietin (TSLP), IL-4 and TGF-β expression was assessed by real-time-PCR in the synovial tissues of RA and OA patients. IL-9, IL-9R, IL-4, TSLP and TGF-β were also investigated by immunohistochemistry. Peripheral CD4(+) T cell subsets were studied by flow cytometry analysis before and after incubation with citrullinated peptides.
RESULTS: IL-9 was overexpressed in RA synovial tissues and correlated with the degree of histological organization of B and T cells in ectopic lymphoid structures. The majority of IL-9-producing cells were identified as CD3(+) cells. Increased mRNA and protein expression of IL-9R, IL-4, TSLP and TGF-β was also observed in RA synovial tissue. Blood peripheral Th9 cells were expanded by citrullinated peptides.
CONCLUSION: These results indicate that Th9 cells and IL-9 were frequently detected in peripheral blood mononuclear cells and synovia of RA patients. A possible pathogenic role for Th9 in RA is discussed.
© The Author 2015. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oup.com.

Entities:  

Keywords:  IL-9; Th9 cells; citrullinated peptide; rheumatoid arthritis

Mesh:

Substances:

Year:  2015        PMID: 26178600     DOI: 10.1093/rheumatology/kev252

Source DB:  PubMed          Journal:  Rheumatology (Oxford)        ISSN: 1462-0324            Impact factor:   7.580


  33 in total

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