Literature DB >> 26170375

Increased levels of neutrophil extracellular trap remnants in the circulation of patients with small vessel vasculitis, but an inverse correlation to anti-neutrophil cytoplasmic antibodies during remission.

Daniel Söderberg1, Tino Kurz2, Atbin Motamedi2, Thomas Hellmark3, Per Eriksson4, Mårten Segelmark5.   

Abstract

OBJECTIVES: Neutrophil extracellular traps (NETs) have been visualized at the site of ANCA-associated vasculitis (AAV) lesions. Increased levels of NET remnants in the circulation have been reported in some AAV patients with active disease. The aim of the present study was to analyse NET remnants in a larger cohort of AAV patients with varying degrees of disease activity and to elucidate possible factors responsible for remnant variation.
METHODS: Levels of NET remnants in the circulation of healthy controls (HCs; n = 31) and AAV patients (n = 93) were determined with ELISA. NET remnants were then correlated with ANCA levels, spontaneous and induced cell death (NETosis/necrosis) in vitro, neutrophil count and corticosteroid therapy.
RESULTS: Patients with active disease showed higher levels of circulating NET remnants compared with patients in remission (P = 0.026) and HCs (P = 0.006). From patients sampled during both remission and active disease, we found increased levels during active disease (P = 0.0010). In remission, ANCA-negative patients had higher levels of NET remnants than ANCA-positive patients and a negative correlation was observed between NET remnants and PR3-ANCA (rs = -0.287, P = 0.048). NET remnants correlated with neutrophil count in HCs (rs = 0.503, P = 0.014) but not in patients during remission. Neutrophils from patients showed enhanced spontaneous cell death (P = 0.043).
CONCLUSION: We found increased levels of circulating NET remnants in patients with active AAV. Furthermore, AAV patients exhibited an increased propensity for spontaneous cell death. NET remnant levels seem to be positively related to disease activity and neutrophil count, but inversely related to ANCA at least during remission.
© The Author 2015. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oup.com.

Entities:  

Keywords:  ANCA; ANCA-associated vasculitis; NET remnants; neutrophil extracellular trap (NET); small vessel vasculitis

Mesh:

Substances:

Year:  2015        PMID: 26170375     DOI: 10.1093/rheumatology/kev217

Source DB:  PubMed          Journal:  Rheumatology (Oxford)        ISSN: 1462-0324            Impact factor:   7.580


  32 in total

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Authors:  Marilina Antonelou; Erik Michaëlsson; Rhys D R Evans; Chun Jing Wang; Scott R Henderson; Lucy S K Walker; Robert John Unwin; Alan D Salama
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Review 4.  Extracellular DNA NET-Works With Dire Consequences for Health.

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Review 5.  Update on ANCA-associated vasculitis: from biomarkers to therapy.

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6.  The potential roles of type I interferon activated neutrophils and neutrophil extracellular traps (NETs) in the pathogenesis of primary Sjögren's syndrome.

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Authors:  Borui Li; Guangtao Li; Xinlei Yang; Zhibo Song; Yu Wang; Zhuoli Zhang
Journal:  Front Immunol       Date:  2022-06-14       Impact factor: 8.786

Review 8.  Disentangling the role of neutrophil extracellular traps in rheumatic diseases.

Authors:  Yaíma L Lightfoot; Mariana J Kaplan
Journal:  Curr Opin Rheumatol       Date:  2017-01       Impact factor: 5.006

9.  Necroptosis controls NET generation and mediates complement activation, endothelial damage, and autoimmune vasculitis.

Authors:  Adrian Schreiber; Anthony Rousselle; Jan Ulrich Becker; Anne von Mässenhausen; Andreas Linkermann; Ralph Kettritz
Journal:  Proc Natl Acad Sci U S A       Date:  2017-10-24       Impact factor: 11.205

Review 10.  Mechanisms of thrombosis in ANCA-associated vasculitis.

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Journal:  Clin Rheumatol       Date:  2021-06-09       Impact factor: 2.980

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