Literature DB >> 2613351

Epidemiology of the allergic diseases: are they really on the increase?

B Wüthrich1.   

Abstract

Epidemiological studies on representative populations clearly demonstrate a significant general increase of atopic diseases during the last decades, mainly for pollinosis. For the manifestation of an atopic disease both allergen exposure, which leads to specific IgE antibody formation, and the presence of additional realization factors are required. The nature of the latter is still partially unknown. Careful interpretations of clinical and experimental observations show that besides indoor and outdoor pollution, increased exposure to allergens through changes in human way of living is an important cofactor responsible for the increased incidence of atopic diseases.

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Year:  1989        PMID： 2613351     DOI： 10.1159/000235067

Source DB:  PubMed          Journal:  Int Arch Allergy Appl Immunol        ISSN： 0020-5915


  23 in total

Review 1.  Family size, infection and atopy: the first decade of the "hygiene hypothesis".

Authors:  D P Strachan
Journal:  Thorax       Date:  2000-08       Impact factor: 9.139

2.  Conformational and linear B-cell epitopes of Asp f 2, a major allergen of Aspergillus fumigatus, bind differently to immunoglobulin E antibody in the sera of allergic bronchopulmonary aspergillosis patients.

Authors:  B Banerjee; P A Greenberger; J N Fink; V P Kurup
Journal:  Infect Immun       Date:  1999-05       Impact factor: 3.441

3.  Nicotine inhibits Fc epsilon RI-induced cysteinyl leukotrienes and cytokine production without affecting mast cell degranulation through alpha 7/alpha 9/alpha 10-nicotinic receptors.

Authors:  Neerad C Mishra; Jules Rir-sima-ah; R Thomas Boyd; Shashi P Singh; Sravanthi Gundavarapu; Raymond J Langley; Seddigheh Razani-Boroujerdi; Mohan L Sopori
Journal:  J Immunol       Date:  2010-05-26       Impact factor: 5.422

4.  Isolation of cDNA encoding a newly identified major allergenic protein of rye-grass pollen: intracellular targeting to the amyloplast.

Authors:  M B Singh; T Hough; P Theerakulpisut; A Avjioglu; S Davies; P M Smith; P Taylor; R J Simpson; L D Ward; J McCluskey
Journal:  Proc Natl Acad Sci U S A       Date:  1991-02-15       Impact factor: 11.205

5.  Trends in prevalence of allergic rhinitis and correlation with pollen counts in Switzerland.

Authors:  Thomas Frei; Ewald Gassner
Journal:  Int J Biometeorol       Date:  2008-08-27       Impact factor: 3.787

6.  Molecular biology and immunology of fungal allergens.

Authors:  V P Kurup; B Banerjee; K J Kelly; J N Fink
Journal:  Indian J Clin Biochem       Date:  2000-08

7.  Antisense-mediated silencing of a gene encoding a major ryegrass pollen allergen.

Authors:  P L Bhalla; I Swoboda; M B Singh
Journal:  Proc Natl Acad Sci U S A       Date:  1999-09-28       Impact factor: 11.205

8.  Systemic mycobacterial infection inhibits antigen-specific immunoglobulin E production, bronchial mucus production and eosinophilic inflammation induced by allergen.

Authors:  X Yang; S Wang; Y Fan; L Zhu
Journal:  Immunology       Date:  1999-11       Impact factor: 7.397

9.  PS80 interferes with the antiallergic effect of Cry-consensus peptide, a novel recombinant peptide for immunotherapy of Japanese cedar pollinosis, at very low concentration through modulation of Th1/Th2 balance.

Authors:  Daisuke Kozutsumi; Masako Tsunematsu; Taketo Yamaji; Rika Murakami; Minehiko Yokoyama; Kohsuke Kino
Journal:  Immunology       Date:  2006-07       Impact factor: 7.397

10.  T cell epitopes of the major fraction of rye grass Lolium perenne (Lol p I) defined using overlapping peptides in vitro and in vivo. I. Isoallergen clone1A.

Authors:  G A Bungy Poor Fard; Y Latchman; S Rodda; M Geysen; I Roitt; J Brostoff
Journal:  Clin Exp Immunol       Date:  1993-10       Impact factor: 4.330
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