Literature DB >> 25991048

Calpain inhibition decreases myocardial apoptosis in a swine model of chronic myocardial ischemia.

Brittany A Potz1, Ashraf A Sabe1, Nassrene Y Elmadhun1, Jun Feng1, Yuhong Liu1, Hunter Mitchell1, Peter Quesenberry1, M Ruhul Abid1, Frank W Sellke2.   

Abstract

INTRODUCTION: Calpain is a family of cysteine proteases that has an important role in the initiation, regulation, and execution of cell death. Our recent studies using a hypercholesterolemic swine model demonstrated that in the setting of the metabolic syndrome, calpain inhibition (CI) improved collateral-dependent perfusion and increased expression of proteins implicated in angiogenesis and vasodilation. In this study, we hypothesized that CI (by MLD28170) would decrease myocardial apoptosis in the same model.
METHODS: Yorkshire swine, all fed a high-cholesterol diet for 4 weeks underwent placement of an ameroid constrictor on the left circumflex coronary artery. Three weeks later, animals received either no drug, termed the high-cholesterol control group (HCC; n = 8); low-dose CI (0.12 mg/kg; LCI, n = 9); or high-dose CI (0.25 mg/kg; HCI, n = 8). The high-cholesterol diet and the CI were continued for 5 weeks, after which the pig was humanely killed and the left ventricular myocardium was harvested and analyzed via terminal deoxynucleotidyl transferase dUTP nick end labeling (TUNEL) staining, oxyblot analysis, and Western blots. Data were analyzed using the Kruskal-Wallis test.
RESULTS: The percentage of apoptotic cells to total cells in ischemic myocardial territory was decreased in the LCI and HCI groups compared with the HCC group as shown by TUNEL staining (P = .018). There was a decrease in proapoptotic proteins, including cleaved caspase 3, caspase 9, cleaved caspase 9, Bax, BAD, p-BAD, and Erk 1/2 (P ≤ .049 each), but no decrease in caspase 3 (P = .737). There was also an increase in antiapoptotic proteins, including BCL-2 and p-BCL2 (P ≤ .025 each). In the ischemic myocardium, several proangiogenic proteins were increased in the LCI and HCI groups compared with the HCC group, including p-AKT, p-eNOS, and eNOS (P ≤ .006 each) but there was no increase in AKT (P = .311). CI decreased tissue oxidative stress in both the LCI and HCI groups compared to the HCC group as shown by oxyblot analysis (P = .021).
CONCLUSION: In the setting of hypercholesterolemia, CI decreases apoptosis and the expression of proteins in proapoptotic signaling pathways. CI also increased expression of proteins implicated in anti apoptotic pathways and improves oxidative stress in ischemic myocardial tissue.
Copyright © 2015 Elsevier Inc. All rights reserved.

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Year:  2015        PMID: 25991048      PMCID: PMC4492842          DOI: 10.1016/j.surg.2015.03.034

Source DB:  PubMed          Journal:  Surgery        ISSN: 0039-6060            Impact factor:   3.982


  20 in total

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2.  Cardiomyocyte degeneration with calpain deficiency reveals a critical role in protein homeostasis.

Authors:  Anita S Galvez; Abhinav Diwan; Amy M Odley; Harvey S Hahn; Hanna Osinska; Jaime G Melendez; Jeffrey Robbins; Roy A Lynch; Yehia Marreez; Gerald W Dorn
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3.  Evidence of endothelial dysfunction in angiographically normal coronary arteries of patients with coronary artery disease.

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4.  Hyperglycemia potentiates collagen-induced platelet activation through mitochondrial superoxide overproduction.

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Review 5.  The pig as a valuable model for testing the effect of resveratrol to prevent cardiovascular disease.

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6.  Metabolic syndrome and risk of cardiovascular disease: a meta-analysis.

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7.  Calpain inhibitors reduce retinal hypoxia in ischemic retinopathy by improving neovascular architecture and functional perfusion.

Authors:  Mien V Hoang; Lois E H Smith; Donald R Senger
Journal:  Biochim Biophys Acta       Date:  2010-09-08

8.  Moderation of calpain activity promotes neovascular integration and lumen formation during VEGF-induced pathological angiogenesis.

Authors:  Mien V Hoang; Janice A Nagy; Joan E B Fox; Donald R Senger
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9.  An ezrin/calpain/PI3K/AMPK/eNOSs1179 signaling cascade mediating VEGF-dependent endothelial nitric oxide production.

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10.  Modulation of coronary vasomotor tone in humans. Progressive endothelial dysfunction with different early stages of coronary atherosclerosis.

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  14 in total

Review 1.  Calpains and Coronary Vascular Disease.

Authors:  Brittany A Potz; Ashraf A Sabe; M Ruhul Abid; Frank W Sellke
Journal:  Circ J       Date:  2015-10-21       Impact factor: 2.993

2.  Diabetes and Cardioplegia.

Authors:  Brittany A Potz; Laura A Scrimgeour; Jun Feng; Frank W Sellke
Journal:  J Nat Sci       Date:  2017-06

3.  Role of Calpain in Pathogenesis of Human Disease Processes.

Authors:  Brittany A Potz; M Ruhul Abid; Frank W Sellke
Journal:  J Nat Sci       Date:  2016

4.  Calpain inhibition modulates glycogen synthase kinase 3β pathways in ischemic myocardium: A proteomic and mechanistic analysis.

Authors:  Brittany A Potz; Ashraf A Sabe; Nassrene Y Elmadhun; Richard T Clements; M Ruhul Abid; Neel R Sodha; Frank W Sellke
Journal:  J Thorac Cardiovasc Surg       Date:  2016-11-16       Impact factor: 5.209

5.  Glycogen synthase kinase 3β inhibition reduces mitochondrial oxidative stress in chronic myocardial ischemia.

Authors:  Brittany A Potz; Laura A Scrimgeour; Sharif A Sabe; Richard T Clements; Neel R Sodha; Frank W Sellke
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6.  Calpain inhibition decreases inflammatory protein expression in vessel walls in a model of chronic myocardial ischemia.

Authors:  Brittany A Potz; Ashraf A Sabe; Nassrene Y Elmadhun; Sharif A Sabe; Benedikt J V Braun; Richard T Clements; Anny Usheva; Frank W Sellke
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Review 7.  Chronic heart failure: Ca(2+), catabolism, and catastrophic cell death.

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Review 9.  Novel molecular targets for coronary angiogenesis and ischemic heart disease.

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10.  Calpain inhibition decreases myocardial fibrosis in chronically ischemic hypercholesterolemic swine.

Authors:  Brittany A Potz; Ashraf A Sabe; Sharif A Sabe; Isabella J Lawandy; M Ruhul Abid; Richard T Clements; Frank W Sellke
Journal:  J Thorac Cardiovasc Surg       Date:  2020-03-29       Impact factor: 5.209

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