Literature DB >> 25877463

Effects of Weipixiao (胃痞消) on Wnt pathway-associated proteins in gastric mucosal epithelial cells from rats with gastric precancerous lesions.

Jin-hao Zeng1, Hua-feng Pan2, You-zhang Liu1, Hai-bo Xu3, Zi-ming Zhao4, Hai-wen Li1, Jin-ling Ren1, Long-hui Chen1, Xia Hu1, Yan Yan1.   

Abstract

OBJECTIVE: To study the effects of Weipixiao (胃痞消, WPX) on Wnt pathway-associated proteins in gastric mucosal epithelial cells from rats with gastric precancerous lesions (GPL).
METHODS: Sprague Dawley rats were randomly divided into control, model, vitacoenzyme (0.2 g·kg(-1)·day(-1)), WPX high-dose (H-WPX, 15 g·kg(-1)·day(-1)), WPX medium-dose (M-WPX, 7.5 g·kg(-1)·day(-1)) and WPX low-dose (L-WPX, 3.75 g·kg(-1)·day(-1)) groups. After successfully establishing the GPL model, the rats were consecutively administered WPX or vitacoenzyme by gastrogavage for 10 weeks. Differential expression of Leucine-rich repeat-containing G-proteincoupled receptor 5 (Lgr5), matrix metalloproteinase-7 (MMP-7), Wnt1, Wnt3a, and β-catenin in gastric mucosal epithelial cells in all groups were immunohistochemically detected, and the images were taken and analyzed semiquantitatively by image pro plus 6.0 software.
RESULTS: Gastric epithelium in the model group showed significantly higher expression levels of Lgr5, MMP-7, Wnt1, Wnt3a and β-catenin than those of the control group(P<0.01). Interestingly, we also observed Lgr5+ cells, which generally located at the base of the gastric glandular unit, migrated to the luminal side of gastric epithelium with GPL. The expression levels of Lgr5, MMP-7, Wnt1, and β-catenin were all down-regulated in the L-WPX group as compared with those of both model and vitacoenzyme groups (P<0.05). A similar, but nonsignificant down-regulation in expression level of Wnt3a was noted in all WPX groups (P>0.05).
CONCLUSION: Our findings suggested that the therapeutic mechanisms of WPX in treating GPL might be related with its inhibitory effects on the expressions of Lgr5, MMP-7, Wnt1, β-catenin and the aberrant activation of Wnt/β-catenin pathway.

Entities:  

Keywords:  Chinese medicine; Lgr5; Weipixiao; Wnt/ß-catenin pathway; Wnt1; Wnt3a; gastric precancerous lesions; matrix metalloproteinase-7; ß-catenin

Mesh:

Substances:

Year:  2015        PMID: 25877463     DOI: 10.1007/s11655-015-2131-4

Source DB:  PubMed          Journal:  Chin J Integr Med        ISSN: 1672-0415            Impact factor:   1.978


  35 in total

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Authors:  T Brabletz; A Jung; S Dag; F Hlubek; T Kirchner
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Journal:  Hepatogastroenterology       Date:  2008 May-Jun

9.  LgR5 expression and cancer stem cell hypothesis: clue to define the true origin of esophageal adenocarcinomas with and without Barrett's esophagus?

Authors:  Burkhard H A von Rahden; Stefan Kircher; Maria Lazariotou; Christoph Reiber; Luisa Stuermer; Christoph Otto; Christoph T Germer; Martin Grimm
Journal:  J Exp Clin Cancer Res       Date:  2011-02-23

10.  Distribution of LGR5+ cells and associated implications during the early stage of gastric tumorigenesis.

Authors:  Bo Gun Jang; Byung Lan Lee; Woo Ho Kim
Journal:  PLoS One       Date:  2013-12-10       Impact factor: 3.240

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