Literature DB >> 25794631

The role of shed placental DNA in the systemic inflammatory syndrome of preeclampsia.

James D R Hartley1, Brian J Ferguson1, Ashley Moffett2.   

Abstract

Preeclampsia is a syndrome occurring only in pregnancy characterized by systemic maternal inflammation and associated with the presence of the placenta. How these 2 aspects of the disease are linked has been the subject of numerous theories and ideas. Recently, there has been increasing interest in DNA shed from the placenta into the maternal circulation as a potential agent initiating the inflammatory response. This review will discuss the current evidence and future directions for placental DNA as the linking factor in preeclampsia in the context of other hypotheses.
Copyright © 2015 Elsevier Inc. All rights reserved.

Entities:  

Keywords:  inflammation; placental DNA; preeclampsia

Mesh：

Substances：

Year:  2015        PMID： 25794631     DOI： 10.1016/j.ajog.2015.03.026

Source DB:  PubMed          Journal:  Am J Obstet Gynecol        ISSN： 0002-9378            Impact factor:   8.661


  11 in total

Review 1.  Preeclampsia and health risks later in life: an immunological link.

Authors:  Shi-Bin Cheng; Surendra Sharma
Journal:  Semin Immunopathol       Date:  2016-06-23       Impact factor: 9.623

Review 2.  Historical evolution of ideas on eclampsia/preeclampsia: A proposed optimistic view of preeclampsia.

Authors:  Pierre-Yves Robillard; Gustaaf Dekker; Gérard Chaouat; Marco Scioscia; Silvia Iacobelli; Thomas C Hulsey
Journal:  J Reprod Immunol       Date:  2017-09-18       Impact factor: 4.054

Review 3.  Maternal-fetal cross talk through cell-free fetal DNA, telomere shortening, microchimerism, and inflammation.

Authors:  Shi-Bin Cheng; Sarah Davis; Surendra Sharma
Journal:  Am J Reprod Immunol       Date:  2018-03-25       Impact factor: 3.886

4.  Circulating DNA and its methylation level in inflammatory bowel disease and related colon cancer.

Authors:  Xuming Bai; Yaqun Zhu; Wangyang Pu; Li Xiao; Kai Li; Chungen Xing; Yong Jin
Journal:  Int J Clin Exp Pathol       Date:  2015-10-01

5.  Curcumin inhibits placental inflammation to ameliorate LPS-induced adverse pregnancy outcomes in mice via upregulation of phosphorylated Akt.

Authors:  Jianjun Zhou; Huishuang Miao; Xiujun Li; Yali Hu; Haixiang Sun; Yayi Hou
Journal:  Inflamm Res       Date:  2016-11-05       Impact factor: 4.575

6.  A Child's HLA-DRB1 genotype increases maternal risk of systemic lupus erythematosus.

Authors:  Giovanna I Cruz; Xiaorong Shao; Hong Quach; Kimberly A Ho; Kirsten Sterba; Janelle A Noble; Nikolaos A Patsopoulos; Michael P Busch; Darrell J Triulzi; Wendy S W Wong; Benjamin D Solomon; John E Niederhuber; Lindsey A Criswell; Lisa F Barcellos
Journal:  J Autoimmun       Date:  2016-07-04       Impact factor: 7.094

7.  The green tea polyphenol EGCG alleviates maternal diabetes-induced neural tube defects by inhibiting DNA hypermethylation.

Authors:  Jianxiang Zhong; Cheng Xu; E Albert Reece; Peixin Yang
Journal:  Am J Obstet Gynecol       Date:  2016-03-12       Impact factor: 8.661

8.  Antiphospholipid antibodies increase the levels of mitochondrial DNA in placental extracellular vesicles: Alarmin-g for preeclampsia.

Authors:  Mancy Tong; Caroline Johansson; Fengyi Xiao; Peter R Stone; Joanna L James; Qi Chen; Lynsey M Cree; Lawrence W Chamley
Journal:  Sci Rep       Date:  2017-11-29       Impact factor: 4.379

9.  Immunological Tolerance, Pregnancy, and Preeclampsia: The Roles of Semen Microbes and the Father.

Authors:  Louise C Kenny; Douglas B Kell
Journal:  Front Med (Lausanne)       Date:  2018-01-04

10.  Endothelial cell leptin receptors, leptin and interleukin-8 in the pathogenesis of preeclampsia: An in-vitro study.

Authors:  Sefa Arlıer
Journal:  Turk J Obstet Gynecol       Date:  2017-12-30
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