Literature DB >> 25740697

Genetic variants in noncoding PIWI-interacting RNA and colorectal cancer risk.

Haiyan Chu1,2, Liping Xia3,4, Xiaonan Qiu1,2, Dongying Gu5, Linjun Zhu6, Jing Jin1,2, Gaoyun Hui1,2, Qiuhan Hua1,2, Mulong Du1,2, Na Tong1,2, Jinfei Chen5, Zhengdong Zhang1,2, Meilin Wang1,2.   

Abstract

BACKGROUND: PIWI-interacting RNAs (piRNAs), which are a novel type of identified small noncoding RNA (ncRNA), play a crucial role in germline development and carcinogenesis.
METHODS: By systematically screening all known piRNAs, the authors identified 7 common single nucleotide polymorphisms (SNPs) in 9 piRNAs. Associations between these selected SNPs and the risk of colorectal cancer (CRC) were detected in a case-control study. A quantitative real-time polymerase chain reaction assay was used to evaluate messenger RNA (mRNA) expression levels of piR-015551 and of the long ncRNA (lncRNA) LNC00964-3 in 88 pairs of tissue samples.
RESULTS: The assay revealed that reference SNP rs11776042 in piR-015551 was significantly associated with a decreased risk of CRC in an additive model (P = .020). However, this protective effect was not significant after correction for multiple comparisons (test for the false discovery rate; P = .140). Furthermore, the authors observed that mRNA expression levels of LNC00964-3 (an lncRNA that included the piR-015551 sequence but not piR-015551) were significantly lower in CRC tissues than in corresponding normal tissues (P = 1.5 × 10(-5) for LNC00964-3; P = .899 for piR-015551). Correlation analysis revealed that piR-015551 expression was positively correlated with expression levels of LNC00964-3 (CRC tissues: r = 0.574, P = 5.13 × 10(-9) ; normal tissues: r = 0.601, P = 5.76 × 10(-10)). Moreover, rs11776042 was not significantly correlated with mRNA expression levels of piR-015551 or LNC00964-3 (all P > .05).
CONCLUSIONS: The current findings reveal the possibility that piR-015551 may be generated from LNC00964-3, which may be involved in the development of CRC.
© 2015 American Cancer Society.

Entities:  

Keywords:  PIWI-interacting RNA; colorectal cancer; genetic variation; long noncoding RNA; molecular epidemiology; susceptibility

Mesh:

Substances:

Year:  2015        PMID: 25740697     DOI: 10.1002/cncr.29314

Source DB:  PubMed          Journal:  Cancer        ISSN: 0008-543X            Impact factor:   6.860


  23 in total

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Authors:  Norhan A Sabbah; Wael M Abdalla; Walid A Mawla; Nagla AbdAlMonem; Amal F Gharib; Ahmed Abdul-Saboor; Abdallah S Abdelazem; Nermin Raafat
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Review 6.  The biogenesis and biological function of PIWI-interacting RNA in cancer.

Authors:  Silu Chen; Shuai Ben; Junyi Xin; Shuwei Li; Rui Zheng; Hao Wang; Lulu Fan; Mulong Du; Zhengdong Zhang; Meilin Wang
Journal:  J Hematol Oncol       Date:  2021-06-12       Impact factor: 17.388

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Journal:  Genes Dis       Date:  2020-10-05

Review 8.  Regulatory Roles of Non-Coding RNAs in Colorectal Cancer.

Authors:  Jun Wang; Yong-Xi Song; Bin Ma; Jia-Jun Wang; Jing-Xu Sun; Xiao-Wan Chen; Jun-Hua Zhao; Yu-Chong Yang; Zhen-Ning Wang
Journal:  Int J Mol Sci       Date:  2015-08-21       Impact factor: 5.923

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Journal:  Arthritis Res Ther       Date:  2015-12-03       Impact factor: 5.156

Review 10.  Long noncoding RNAs in the progression, metastasis, and prognosis of osteosarcoma.

Authors:  Zuozhang Yang; Xiaojuan Li; Yihao Yang; Zewei He; Xin Qu; Ya Zhang
Journal:  Cell Death Dis       Date:  2016-09-29       Impact factor: 8.469

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