Literature DB >> 25555639

Apoptosis in chronic tonsillitis and tonsillar hypertrophy.

Merih Önal1, Taner Yılmaz2, Elif Bilgiç3, Sevda Fatma Müftüoğlu4, Oğuz Kuşçu5, Rıza Önder Günaydın6.   

Abstract

OBJECTIVE: Chronic tonsillitis is the persistent inflammation of the tonsillar tissue that occurs due to recurrent, acute or subclinical infection. The recurrent and chronic inflammation of palatine tonsils sometimes results in hypertrophy. Apoptosis provides an important balance between lymphocytes in tonsillar lymphoid tissue. The aim of this study is to investigate the apoptosis in tonsillar diseases.
METHODS: 43 patients with chronic tonsilitis and tonsillar hypertrophy underwent tonsillectomy. The specimens were examined immunohistochemically for apoptosis. Tonsils were assembled into groups according to their size. Specimens were compared for their apoptotic cell count.
RESULTS: The apoptosis difference between the tonsil size groups is not statistically significant (p>0.05). However, when the study group was divided into two at age 6, the difference was not statistically significant for patients at and below 6 years of age; but, the difference was statistically significant for patients above 6 years of age (p<0.05). The comparison of apoptosis in microcompartments of tonsil tissue (intrafollicular, interfollicular, subepithelial and intraepithelial) between tonsil size stages and between chronic tonsillitis and tonsillar hypertrophy groups revealed no statistical significance (p>0.05). There was a statistically significant positive correlation between intrafollicular and interfollicular, interfollicular and intraepithelial &amp; subepithelial and intraepithelial areas (p<0.05).
CONCLUSIONS: In the light of these findings, it was concluded that apoptosis played a role in the tonsillar hypertrophy and atrophy. Apoptosis functioned to balance lymphocyte proliferation in tonsil tissue. The association of apoptosis with tonsillar hypertrophy seemed to be age-dependent.
Copyright © 2014 Elsevier Ireland Ltd. All rights reserved.

Entities:  

Keywords:  Apoptosis; In situ nick-end labeling; Palatine tonsil; Tonsillitis

Mesh:

Year:  2014        PMID: 25555639     DOI: 10.1016/j.ijporl.2014.12.005

Source DB:  PubMed          Journal:  Int J Pediatr Otorhinolaryngol        ISSN: 0165-5876            Impact factor:   1.675


  5 in total

1.  Extra-Esophageal Pepsin from Stomach Refluxate Promoted Tonsil Hypertrophy.

Authors:  Jin Hyun Kim; Han-Sin Jeong; Kyung Mi Kim; Ye Jin Lee; Myeong Hee Jung; Jung Je Park; Jin Pyeong Kim; Seung Hoon Woo
Journal:  PLoS One       Date:  2016-04-08       Impact factor: 3.240

2.  Effects of pepsin and pepstatin on reflux tonsil hypertrophy in vitro.

Authors:  Jin Hyun Kim; Si Jung Jang; Jeong Won Yun; Myeong Hee Jung; Seung Hoon Woo
Journal:  PLoS One       Date:  2018-11-08       Impact factor: 3.240

Review 3.  Anatomy and physiology of the palatine tonsils, adenoids, and lingual tonsils.

Authors:  Alexandra Arambula; Jason R Brown; Laura Neff
Journal:  World J Otorhinolaryngol Head Neck Surg       Date:  2021-06-27

4.  Relationship of endoplasmic reticulum stress with the etiopathogenesis of chronic tonsillitis and tonsillar hypertrophy in pediatric patients: a prospective, parallel-group study.

Authors:  Merih Onal; Nadir Kocak; Fahrettin Duymus; Mete Kaan Bozkurt; Cagdas Elsurer; Omer Erdur; Ozkan Onal
Journal:  Mol Biol Rep       Date:  2021-07-22       Impact factor: 2.316

5.  Expression and subcellular localisation of AID and APOBEC3 in adenoid and palatine tonsils.

Authors:  Noriko Seishima; Satoru Kondo; Kousho Wakae; Naohiro Wakisaka; Eiji Kobayashi; Makoto Kano; Makiko Moriyama-Kita; Yosuke Nakanishi; Kazuhira Endo; Tomoko Imoto; Kazuya Ishikawa; Hisashi Sugimoto; Miyako Hatano; Takayoshi Ueno; Miki Koura; Koichi Kitamura; Masamichi Muramatsu; Tomokazu Yoshizaki
Journal:  Sci Rep       Date:  2018-01-17       Impact factor: 4.379

  5 in total

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