Nicole A Short1, Nicholas P Allan1, Amanda M Raines1, Norman B Schmidt2. 1. Department of Psychology, Florida State University, Tallahassee, FL, USA. 2. Department of Psychology, Florida State University, Tallahassee, FL, USA. Electronic address: schmidt@psy.fsu.edu.
Abstract
OBJECTIVE: Recent work suggests a link between a transdiagnostic vulnerability factor, anxiety sensitivity (AS), and sleep disturbance. Although research has indicated that AS is malleable through brief interventions, no studies have yet examined whether interventions targeting AS will reduce symptoms of insomnia. Considering this gap in previous research, the current study tested the direct and indirect effects of a brief, computerized intervention targeting AS on self-reported insomnia symptoms. METHODS:Community participants (N = 97) were randomized into either the AS intervention (consisting of psychoeducation and interoceptive exposure) or a health information control condition, and they were assessed at baseline and at 1-month follow-up. RESULTS: Findings indicated that symptoms of insomnia were related to AS and its subfactors at baseline and follow-up. Moreover, there was an indirect effect of the treatment on insomnia symptoms through AS and its subfactors, which held after covarying for baseline symptoms of anxiety and depression. CONCLUSIONS: These preliminary findings suggest that targeting AS may be a brief and effective way to reduce symptoms of insomnia, but it should be replicated in a clinical sample of individuals with a diagnosis of insomnia disorder.
RCT Entities:
OBJECTIVE: Recent work suggests a link between a transdiagnostic vulnerability factor, anxiety sensitivity (AS), and sleep disturbance. Although research has indicated that AS is malleable through brief interventions, no studies have yet examined whether interventions targeting AS will reduce symptoms of insomnia. Considering this gap in previous research, the current study tested the direct and indirect effects of a brief, computerized intervention targeting AS on self-reported insomnia symptoms. METHODS: Community participants (N = 97) were randomized into either the AS intervention (consisting of psychoeducation and interoceptive exposure) or a health information control condition, and they were assessed at baseline and at 1-month follow-up. RESULTS: Findings indicated that symptoms of insomnia were related to AS and its subfactors at baseline and follow-up. Moreover, there was an indirect effect of the treatment on insomnia symptoms through AS and its subfactors, which held after covarying for baseline symptoms of anxiety and depression. CONCLUSIONS: These preliminary findings suggest that targeting AS may be a brief and effective way to reduce symptoms of insomnia, but it should be replicated in a clinical sample of individuals with a diagnosis of insomnia disorder.
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