Literature DB >> 25362193

Tracking a tuberculosis outbreak over 21 years: strain-specific single-nucleotide polymorphism typing combined with targeted whole-genome sequencing.

David Stucki1, Marie Ballif2, Thomas Bodmer3, Mireia Coscolla1, Anne-Marie Maurer4, Sara Droz5, Christa Butz6, Sonia Borrell1, Christel Längle5, Julia Feldmann1, Hansjakob Furrer7, Carlo Mordasini6, Peter Helbling8, Hans L Rieder9, Matthias Egger10, Sébastien Gagneux1, Lukas Fenner11.   

Abstract

BACKGROUND: Whole-genome sequencing (WGS) is increasingly used in molecular-epidemiological investigations of bacterial pathogens, despite cost- and time-intensive analyses. We combined strain-specific single-nucleotide polymorphism (SNP) typing and targeted WGS to investigate a tuberculosis cluster spanning 21 years in Bern, Switzerland.
METHODS: On the basis of genome sequences of 3 historical outbreak Mycobacterium tuberculosis isolates, we developed a strain-specific SNP-typing assay to identify further cases. We screened 1642 patient isolates and performed WGS on all identified cluster isolates. We extracted SNPs to construct genomic networks. Clinical and social data were retrospectively collected.
RESULTS: We identified 68 patients associated with the outbreak strain. Most received a tuberculosis diagnosis in 1991-1995, but cases were observed until 2011. Two thirds were homeless and/or substance abusers. Targeted WGS revealed 133 variable SNP positions among outbreak isolates. Genomic network analyses suggested a single origin of the outbreak, with subsequent division into 3 subclusters. Isolates from patients with confirmed epidemiological links differed by 0-11 SNPs.
CONCLUSIONS: Strain-specific SNP genotyping allowed rapid and inexpensive identification of M. tuberculosis outbreak isolates in a population-based strain collection. Subsequent targeted WGS provided detailed insights into transmission dynamics. This combined approach could be applied to track bacterial pathogens in real time and at high resolution.
© The Author 2014. Published by Oxford University Press on behalf of the Infectious Diseases Society of America. All rights reserved. For Permissions, please e-mail: journals.permissions@oup.com.

Entities:  

Keywords:  genomic epidemiology; outbreak; screening; tuberculosis; whole genome sequencing

Mesh:

Substances:

Year:  2014        PMID: 25362193      PMCID: PMC4447836          DOI: 10.1093/infdis/jiu601

Source DB:  PubMed          Journal:  J Infect Dis        ISSN: 0022-1899            Impact factor:   5.226


  31 in total

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Journal:  Lancet Infect Dis       Date:  2012-11-15       Impact factor: 25.071

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8.  Nonsynonymous Polymorphism Counts in Bacterial Genomes: a Comparative Examination.

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9.  Fitness-compensatory mutations facilitate the spread of drug-resistant F15/LAM4/KZN and F28 Mycobacterium tuberculosis strains in KwaZulu-Natal, South Africa.

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