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Literature DB >> 25350065

Bone: Key role for S1P in bone remodelling.

David Holmes.

Abstract

Entities: Gene

Mesh：

Substances：

Year: 2014 PMID： 25350065 DOI： 10.1038/nrendo.2014.190

Source DB: PubMed Journal: Nat Rev Endocrinol ISSN： 1759-5029 Impact factor: 43.330

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1 in total

1. Calcitonin controls bone formation by inhibiting the release of sphingosine 1-phosphate from osteoclasts.

Authors: Johannes Keller; Philip Catala-Lehnen; Antje K Huebner; Anke Jeschke; Timo Heckt; Anja Lueth; Matthias Krause; Till Koehne; Joachim Albers; Jochen Schulze; Sarah Schilling; Michael Haberland; Hannah Denninger; Mona Neven; Irm Hermans-Borgmeyer; Thomas Streichert; Stefan Breer; Florian Barvencik; Bodo Levkau; Birgit Rathkolb; Eckhard Wolf; Julia Calzada-Wack; Frauke Neff; Valerie Gailus-Durner; Helmut Fuchs; Martin Hrabĕ de Angelis; Susanne Klutmann; Elena Tsourdi; Lorenz C Hofbauer; Burkhard Kleuser; Jerold Chun; Thorsten Schinke; Michael Amling
Journal: Nat Commun Date: 2014-10-21 Impact factor: 14.919

1 in total

3 in total

Bone: Key role for S1P in bone remodelling.

1. Calcitonin controls bone formation by inhibiting the release of sphingosine 1-phosphate from osteoclasts.

1. De novo Synthesis of Sphingolipids Is Defective in Experimental Models of Huntington's Disease.

Review 2. Sphingolipid Metabolism: A New Therapeutic Opportunity for Brain Degenerative Disorders.

3. LC-MS-Based Untargeted Metabolomics Reveals Early Biomarkers in STZ-Induced Diabetic Rats With Cognitive Impairment.