Literature DB >> 25139874

Association of interleukin-6 with the progression of carotid atherosclerosis: a 9-year follow-up study.

Shuhei Okazaki1, Manabu Sakaguchi2, Kaori Miwa2, Shigetaka Furukado2, Hiroshi Yamagami2, Yoshiki Yagita2, Hideki Mochizuki2, Kazuo Kitagawa1.   

Abstract

BACKGROUND AND
PURPOSE: Limited information is available on the long-term effects of interleukin-6 (IL-6) on systemic atherosclerosis. The purpose of the present study was to clarify the relationship between chronic elevation of IL-6 and the long-term progression of carotid atherosclerosis.
METHODS: We prospectively evaluated 210 patients with ≥1 vascular risk factors for 9.0±1.0 years. Carotid mean-maximal intima-media thickness (mmIMT), the serum high-sensitivity C-reactive protein (hs-CRP) level, and the serum IL-6 level were measured at baseline and every 3 years. The associations between the progression of mmIMT and the long-term average levels of hs-CRP and IL-6 were analyzed.
RESULTS: Carotid mmIMT increased throughout the study period (0.031±0.026 mm/y). Baseline mmIMT was significantly associated with baseline hs-CRP (P=0.002) and baseline IL-6 (P<0.001) levels. Progression of mmIMT was positively correlated with average hs-CRP (P=0.001) and average IL-6 (P<0.001) levels. When adjusted for age, sex, traditional risk factors, and baseline mmIMT, mmIMT progression remained significantly associated only with the average IL-6 level (standardized β=0.17; P=0.02), but not with the average hs-CRP level (standardized β=0.10; P=0.18).
CONCLUSIONS: Chronic elevation of serum IL-6 was associated with the progression of atherosclerosis in patients with vascular risk factors. IL-6 could be used as a quantitative marker and a potential therapeutic target for accelerated atherosclerosis.
© 2014 American Heart Association, Inc.

Entities:  

Keywords:  atherosclerosis; follow-up studies; inflammation; interleukins; risk factors

Mesh:

Substances:

Year:  2014        PMID: 25139874     DOI: 10.1161/STROKEAHA.114.005991

Source DB:  PubMed          Journal:  Stroke        ISSN: 0039-2499            Impact factor:   7.914


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