| Literature DB >> 24726269 |
Nathalie Douet-Guilbert1, Corinne Tous2, Glen Le Flahec2, Clément Bovo3, Marie-Josée Le Bris2, Audrey Basinko2, Frédéric Morel1, Marc De Braekeleer4.
Abstract
Structural abnormalities of chromosome 7q have been regularly reported in chronic B-cell lymphoproliferative disorders. They include chromosomal translocations involving 7q21, leading to overexpression of the CDK6 gene. Three different translocations, t(7;14)(q21;q32), t(7;22)(q21;q11), and t(2;7)(p11;q21), leading to the juxtaposition of the CDK6 gene with a immunoglobulin gene enhancer during B-cell differentiation, have been described. In the past 2 years, we identified three patients with lymphoproliferative malignancy associated with a t(2;7)(p11;q21). Fluorescent in situ hybridization using an IGK probe and a library of bacterial artificial chromosome (BAC) clones located in bands 7q21.2 and 7q21.3, containing CDK6, revealed that the telomeric part of the IGK probe was translocated on the der(7) within a 51-kb region upstream of the transcriptional start site of CDK6. A total of 23 patients with indolent B-cell lymphoproliferative disorders and juxtaposition of the IG and CDK6 genes, including 20 with IGK and CDK6 juxtaposition, have been reported thus far. This rearrangement leads to the overexpression of CDK6, which encodes a cyclin-dependent protein kinase involved in cell cycle G1 phase progression and G1/S transition.Entities:
Keywords: B-cell lymphoproliferative malignancy; CDK6/IGK; chromosomal abnormality; t(2;7)(p11;q21)
Mesh:
Year: 2014 PMID: 24726269 DOI: 10.1016/j.cancergen.2014.02.009
Source DB: PubMed Journal: Cancer Genet