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Literature DB >> 24699084

RPA puts the brakes on MMEJ.

Mitch McVey¹.

Abstract

Mesh：

Substances：

Year: 2014 PMID： 24699084 DOI： 10.1038/nsmb.2802

Source DB: PubMed Journal: Nat Struct Mol Biol ISSN： 1545-9985 Impact factor: 15.369

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20 in total

1. Saccharomyces cerevisiae Sae2- and Tel1-dependent single-strand DNA formation at DNA break promotes microhomology-mediated end joining.

Authors: Kihoon Lee; Sang Eun Lee
Journal: Genetics Date: 2007-06-11 Impact factor: 4.562

2. A proteome-wide analysis of kinase-substrate network in the DNA damage response.

Authors: Sheng-hong Chen; Claudio P Albuquerque; Jason Liang; Raymond T Suhandynata; Huilin Zhou
Journal: J Biol Chem Date: 2010-02-27 Impact factor: 5.157

3. DNA annealing by RAD52 protein is stimulated by specific interaction with the complex of replication protein A and single-stranded DNA.

Authors: T Sugiyama; J H New; S C Kowalczykowski
Journal: Proc Natl Acad Sci U S A Date: 1998-05-26 Impact factor: 11.205

4. An essential role for CtIP in chromosomal translocation formation through an alternative end-joining pathway.

Authors: Yu Zhang; Maria Jasin
Journal: Nat Struct Mol Biol Date: 2010-12-05 Impact factor: 15.369

5. Ku regulates the non-homologous end joining pathway choice of DNA double-strand break repair in human somatic cells.

Authors: Farjana Fattah; Eu Han Lee; Natalie Weisensel; Yongbao Wang; Natalie Lichter; Eric A Hendrickson
Journal: PLoS Genet Date: 2010-02-26 Impact factor: 5.917

6. The Mre11/Rad50/Nbs1 complex functions in resection-based DNA end joining in Xenopus laevis.

Authors: Elaine M Taylor; Sophie M Cecillon; Antonio Bonis; J Ross Chapman; Lawrence F Povirk; Howard D Lindsay
Journal: Nucleic Acids Res Date: 2009-11-05 Impact factor: 16.971

7. An allele of RFA1 suppresses RAD52-dependent double-strand break repair in Saccharomyces cerevisiae.

Authors: J Smith; R Rothstein
Journal: Genetics Date: 1999-02 Impact factor: 4.562

8. Yeast Mre11 and Rad1 proteins define a Ku-independent mechanism to repair double-strand breaks lacking overlapping end sequences.

Authors: Jia-Lin Ma; Eun Mi Kim; James E Haber; Sang Eun Lee
Journal: Mol Cell Biol Date: 2003-12 Impact factor: 4.272

9. A mutation in the gene encoding the Saccharomyces cerevisiae single-stranded DNA-binding protein Rfa1 stimulates a RAD52-independent pathway for direct-repeat recombination.

Authors: J Smith; R Rothstein
Journal: Mol Cell Biol Date: 1995-03 Impact factor: 4.272

RPA puts the brakes on MMEJ.

1. Saccharomyces cerevisiae Sae2- and Tel1-dependent single-strand DNA formation at DNA break promotes microhomology-mediated end joining.

2. A proteome-wide analysis of kinase-substrate network in the DNA damage response.

3. DNA annealing by RAD52 protein is stimulated by specific interaction with the complex of replication protein A and single-stranded DNA.

4. An essential role for CtIP in chromosomal translocation formation through an alternative end-joining pathway.

5. Ku regulates the non-homologous end joining pathway choice of DNA double-strand break repair in human somatic cells.

6. The Mre11/Rad50/Nbs1 complex functions in resection-based DNA end joining in Xenopus laevis.

7. An allele of RFA1 suppresses RAD52-dependent double-strand break repair in Saccharomyces cerevisiae.

8. Yeast Mre11 and Rad1 proteins define a Ku-independent mechanism to repair double-strand breaks lacking overlapping end sequences.

9. A mutation in the gene encoding the Saccharomyces cerevisiae single-stranded DNA-binding protein Rfa1 stimulates a RAD52-independent pathway for direct-repeat recombination.

10. Alternative end-joining mechanisms: a historical perspective.

Review 1. Risky business: Microhomology-mediated end joining.

2. MMEJ-assisted gene knock-in using TALENs and CRISPR-Cas9 with the PITCh systems.

3. Microhomology Selection for Microhomology Mediated End Joining in Saccharomyces cerevisiae.

4. RB Regulates DNA Double Strand Break Repair Pathway Choice by Mediating CtIP Dependent End Resection.

5. Gene manipulation in the Mucorales fungus Rhizopus oryzae using TALENs with exonuclease overexpression.

6. A role for human homologous recombination factors in suppressing microhomology-mediated end joining.