Literature DB >> 24572674

Association of CAV1/CAV2 genomic variants with primary open-angle glaucoma overall and by gender and pattern of visual field loss.

Stephanie J Loomis1, Jae H Kang2, Robert N Weinreb3, Brian L Yaspan4, Jessica N Cooke Bailey5, Douglas Gaasterland6, Terry Gaasterland7, Richard K Lee8, Paul R Lichter9, Donald L Budenz10, Yutao Liu11, Tony Realini12, David S Friedman13, Catherine A McCarty14, Sayoko E Moroi9, Lana Olson5, Joel S Schuman15, Kuldev Singh16, Douglas Vollrath17, Gadi Wollstein15, Donald J Zack13, Murray Brilliant18, Arthur J Sit19, William G Christen2, John Fingert20, Peter Kraft21, Kang Zhang3, R Rand Allingham22, Margaret A Pericak-Vance8, Julia E Richards9, Michael A Hauser11, Jonathan L Haines5, Louis R Pasquale23, Janey L Wiggs24.   

Abstract

PURPOSE: The CAV1/CAV2 (caveolin 1 and caveolin 2) genomic region previously was associated with primary open-angle glaucoma (POAG), although replication among independent studies has been variable. The aim of this study was to assess the association between CAV1/CAV2 single nucleotide polymorphisms (SNPs) and POAG in a large case-control dataset and to explore associations by gender and pattern of visual field (VF) loss further.
DESIGN: Case-control study. PARTICIPANTS: We analyzed 2 large POAG data sets: the Glaucoma Genes and Environment (GLAUGEN) study (976 cases, 1140 controls) and the National Eye Institute Glaucoma Human Genetics Collaboration (NEIGHBOR) consortium (2132 cases, 2290 controls).
METHODS: We studied the association between 70 SNPs located within the CAV1/CAV2 genomic region in the GLAUGEN and NEIGHBOR studies, both genotyped on the Illumina Human 660WQuadv1C BeadChip array and imputed with the Markov Chain Haplotyping algorithm using the HapMap 3 reference panel. We used logistic regression models of POAG in the overall population and separated by gender, as well as by POAG subtypes defined by type of VF defect (peripheral or paracentral). Results from GLAUGEN and NEIGHBOR were meta-analyzed, and a Bonferroni-corrected significance level of 7.7 × 10(-4) was used to account for multiple comparisons. MAIN OUTCOME MEASURES: Overall POAG, overall POAG by gender, and POAG subtypes defined by pattern of early VF loss.
RESULTS: We found significant associations between 10 CAV1/CAV2 SNPs and POAG (top SNP, rs4236601; pooled P = 2.61 × 10(-7)). Of these, 9 were significant only in women (top SNP, rs4236601; pooled P = 1.59 × 10(-5)). Five of the 10 CAV1/CAV2 SNPs were associated with POAG with early paracentral VF (top SNP, rs17588172; pooled P = 1.07 × 10(-4)), and none of the 10 were associated with POAG with peripheral VF loss only or POAG among men.
CONCLUSIONS: CAV1/CAV2 SNPs were associated significantly with POAG overall, particularly among women. Furthermore, we found an association between CAV1/CAV2 SNPs and POAG with paracentral VF defects. These data support a role for caveolin 1, caveolin 2, or both in POAG and suggest that the caveolins particularly may affect POAG pathogenesis in women and in patients with early paracentral VF defects.
Copyright © 2014 American Academy of Ophthalmology. Published by Elsevier Inc. All rights reserved.

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Year:  2013        PMID: 24572674      PMCID: PMC3937766          DOI: 10.1016/j.ophtha.2013.09.012

Source DB:  PubMed          Journal:  Ophthalmology        ISSN: 0161-6420            Impact factor:   12.079


  42 in total

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2.  Transforming growth factor-beta receptors localize to caveolae and regulate endothelial nitric oxide synthase in normal human endothelial cells.

Authors:  Eric A Schwartz; Eve Reaven; James N Topper; Philip S Tsao
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Authors:  M Motolko; S M Drance; G R Douglas
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4.  Genome-wide association study identifies susceptibility loci for open angle glaucoma at TMCO1 and CDKN2B-AS1.

Authors:  Kathryn P Burdon; Stuart Macgregor; Alex W Hewitt; Shiwani Sharma; Glyn Chidlow; Richard A Mills; Patrick Danoy; Robert Casson; Ananth C Viswanathan; Jimmy Z Liu; John Landers; Anjali K Henders; John Wood; Emmanuelle Souzeau; April Crawford; Paul Leo; Jie Jin Wang; Elena Rochtchina; Dale R Nyholt; Nicholas G Martin; Grant W Montgomery; Paul Mitchell; Matthew A Brown; David A Mackey; Jamie E Craig
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5.  Clinical implications of old and new genes for open-angle glaucoma.

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  45 in total

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2.  Caveolin-1 increases proinflammatory chemoattractants and blood-retinal barrier breakdown but decreases leukocyte recruitment in inflammation.

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5.  Vascular tone pathway polymorphisms in relation to primary open-angle glaucoma.

Authors:  J H Kang; S J Loomis; B L Yaspan; J C Bailey; R N Weinreb; R K Lee; P R Lichter; D L Budenz; Y Liu; T Realini; D Gaasterland; T Gaasterland; D S Friedman; C A McCarty; S E Moroi; L Olson; J S Schuman; K Singh; D Vollrath; G Wollstein; D J Zack; M Brilliant; A J Sit; W G Christen; J Fingert; J P Forman; E S Buys; P Kraft; K Zhang; R R Allingham; M A Pericak-Vance; J E Richards; M A Hauser; J L Haines; J L Wiggs; L R Pasquale
Journal:  Eye (Lond)       Date:  2014-03-07       Impact factor: 3.775

Review 6.  Common and rare genetic risk factors for glaucoma.

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Review 7.  Caveolins and caveolae in ocular physiology and pathophysiology.

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8.  The African Descent and Glaucoma Evaluation Study (ADAGES) III: Contribution of Genotype to Glaucoma Phenotype in African Americans: Study Design and Baseline Data.

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Journal:  Ophthalmology       Date:  2018-02-01       Impact factor: 12.079

9.  Comparison of Risk Factor Profiles for Primary Open-Angle Glaucoma Subtypes Defined by Pattern of Visual Field Loss: A Prospective Study.

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10.  Developments in Ocular Genetics: 2013 Annual Review.

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