Literature DB >> 24522888

High Sam68 expression predicts poor prognosis in non-small cell lung cancer.

Z Zhang1, Y Xu, N Sun, M Zhang, J Xie, Z Jiang.   

Abstract

BACKGROUND: The nuclear protein Sam68 has been implicated in the oncogenesis and tumor growth. The aim of this study was to explore the clinical value of Sam68 in patients with non-small cell lung cancer (NSCLC).
METHODS: We examined Sam68 expression in 50 NSCLC tissues and matched adjacent noncancerous tissues by quantitative RT-PCR (qRT-PCR) and Western blotting. Furthermore, the Sam68 protein expression was analyzed by immunohistochemistry in 208 NSCLC samples. Kaplan-Meier method and multivariate Cox regression model were used to evaluate the prognostic value of nuclear Sam68 expression in NSCLC for disease survival.
RESULTS: The expression of Sam68 was significantly elevated in NSCLC tissues as compared with adjacent non-cancerous tissues (P < 0.01). The high expression of Sam68 in NSCLC was significantly correlated with lymph node metastasis and tumor TNM stage. Kaplan-Meier survival analysis revealed that high expression of Sam68 correlated with poor prognosis of NSCLC patients (P < 0.01). Multivariate analysis showed that Sam68 expression was an independent prognostic marker for overall survival of NSCLC patients (HR 2.73, 95 % CI 1.549-4.315, P = 0.002).
CONCLUSION: Our results suggest that high Sam68 expression predicts poor prognosis of NSCLC patients, and Sam68 may be potentially a prognostic biomarker for NSCLC.

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Year:  2014        PMID: 24522888     DOI: 10.1007/s12094-014-1160-3

Source DB:  PubMed          Journal:  Clin Transl Oncol        ISSN: 1699-048X            Impact factor:   3.405


  25 in total

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2.  Signal-dependent regulation of splicing via phosphorylation of Sam68.

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Review 4.  Staging of non-small cell lung cancer (NSCLC): a review.

Authors:  S Tsim; C A O'Dowd; R Milroy; S Davidson
Journal:  Respir Med       Date:  2010-09-15       Impact factor: 3.415

Review 5.  Pharmacogenomics of platinum-based chemotherapy in NSCLC.

Authors:  Michelle A T Hildebrandt; Jian Gu; Xifeng Wu
Journal:  Expert Opin Drug Metab Toxicol       Date:  2009-07       Impact factor: 4.481

6.  Sam68 regulates EMT through alternative splicing-activated nonsense-mediated mRNA decay of the SF2/ASF proto-oncogene.

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Review 7.  Regulation of gene expression by the RNA-binding protein Sam68 in cancer.

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2.  Clinical significance of Sam68 expression in endometrial carcinoma.

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Review 7.  The Role of RNA Splicing Factors in Cancer: Regulation of Viral and Human Gene Expression in Human Papillomavirus-Related Cervical Cancer.

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8.  A comprehensive study on genome-wide coexpression network of KHDRBS1/Sam68 reveals its cancer and patient-specific association.

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10.  The p53 status can influence the role of Sam68 in tumorigenesis.

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