Literature DB >> 24332005

Histologic study of a human immature permanent premolar with chronic apical abscess after revascularization/revitalization.

Patricia Becerra1, Domenico Ricucci2, Simona Loghin3, Jennifer L Gibbs4, Louis M Lin4.   

Abstract

INTRODUCTION: Histologic studies of teeth from animal models of revascularization/revitalization are available; however, specimens from human studies are lacking. The nature of tissues formed in the canal of human revascularized/revitalized teeth was not well established.
METHODS: An immature mandibular premolar with infected necrotic pulp and a chronic apical abscess was treated with revascularization/revitalization procedures. At both the 18-month and 2-year follow-up visits, radiographic examination showed complete resolution of the periapical lesion, narrowing of the root apex without root lengthening, and minimal thickening of the canal walls. The revascularized/revitalized tooth was removed because of orthodontic treatment and processed for histologic examination.
RESULTS: The large canal space of revascularized/revitalized tooth was not empty and filled with fibrous connective tissue. The apical closure was caused by cementum deposition without dentin. Some cementum-like tissue was formed on the canal dentin walls. Inflammatory cells were observed in the coronal and middle third of revascularized/revitalized tissue.
CONCLUSIONS: In the present case, the tissue formed in the canal of a human revascularized/revitalized tooth was soft connective tissue similar to that in the periodontal ligament and cementum-like or bone-like hard tissue, which is comparable with the histology observed in the canals of teeth from animal models of revascularization/revitalization.
Copyright © 2014 American Association of Endodontists. Published by Elsevier Inc. All rights reserved.

Entities:  

Keywords:  Bone-like tissue; cementum-like tissue; chronic apical abscess; human immature permanent tooth; revascularization/revitalization

Mesh:

Substances:

Year:  2013        PMID: 24332005     DOI: 10.1016/j.joen.2013.07.017

Source DB:  PubMed          Journal:  J Endod        ISSN: 0099-2399            Impact factor:   4.171


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