Literature DB >> 24311643

Phase II study of everolimus in children and adults with neurofibromatosis type 2 and progressive vestibular schwannomas.

Matthias A Karajannis1, Geneviève Legault, Mari Hagiwara, Filippo G Giancotti, Alexander Filatov, Anna Derman, Tsivia Hochman, Judith D Goldberg, Emilio Vega, Jeffrey H Wisoff, John G Golfinos, Amanda Merkelson, J Thomas Roland, Jeffrey C Allen.   

Abstract

BACKGROUND: Activation of the mammalian target of rapamycin (mTOR) signaling pathway is thought to be a key driver of tumor growth in Merlin (NF2)-deficient tumors. Everolimus is an oral inhibitor of mTOR complex 1 (mTORC1) with antitumor activity in a variety of cancers.
METHODS: We conducted a single-institution, prospective, 2-stage, open-label phase II study to estimate the response rate to everolimus in neurofibromatosis type 2 (NF2) patients with progressive vestibular schwannoma (VS). Ten eligible patients were enrolled, including 2 pediatric patients. Everolimus was administered at a daily dose of 10 mg (adults) or 5 mg/m(2)/day (children <18 y) orally in continuous 28-day courses, for up to 12 courses. Response was assessed every 3 months with MRI, using 3-dimensional volumetric tumor analysis, and audiograms. Nine patients were evaluable for the primary response, defined as ≥15% decrease in VS volume. Hearing response was evaluable as a secondary endpoint in 8 patients.
RESULTS: None of the 9 patients with evaluable disease experienced a clinical or MRI response. No objective imaging or hearing responses were observed in stage 1 of the trial, and the study was closed according to predefined stopping rules.
CONCLUSION: Everolimus is ineffective for the treatment of progressive VS in NF2 patients. We are currently conducting a pharmacokinetic/pharmacodynamic ("phase 0") study of everolimus in presurgical VS patients to elucidate the biological basis for apparent treatment resistance to mTORC1 inhibition in these tumors.

Entities:  

Keywords:  everolimus; mammalian target of rapamycin; neurofibromatosis type 2; phase II trial; vestibular schwannoma.

Mesh:

Substances:

Year:  2013        PMID: 24311643      PMCID: PMC3895376          DOI: 10.1093/neuonc/not150

Source DB:  PubMed          Journal:  Neuro Oncol        ISSN: 1522-8517            Impact factor:   12.300


  33 in total

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2.  Regulation of mTOR complex 2 signaling in neurofibromatosis 2-deficient target cell types.

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6.  Phase II trial of lapatinib in adult and pediatric patients with neurofibromatosis type 2 and progressive vestibular schwannomas.

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  36 in total

1.  Phase II study of mTORC1 inhibition by everolimus in neurofibromatosis type 2 patients with growing vestibular schwannomas.

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6.  Preclinical assessment of MEK1/2 inhibitors for neurofibromatosis type 2-associated schwannomas reveals differences in efficacy and drug resistance development.

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7.  Merlin status regulates p75(NTR) expression and apoptotic signaling in Schwann cells following nerve injury.

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8.  Bevacizumab decreases vestibular schwannomas growth rate in children and teenagers with neurofibromatosis type 2.

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9.  Effects of Neurod1 Expression on Mouse and Human Schwannoma Cells.

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Review 10.  Neurofibromatosis-related tumors: emerging biology and therapies.

Authors:  Matthias A Karajannis; Rosalie E Ferner
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