Literature DB >> 24267971

Is there pseudoprogression in secondary glioblastomas?

Tareq A Juratli1, Kay Engellandt, Tim Lautenschlaeger, Kathrin D Geiger, Rüdiger von Kummer, Jana Cerhova, Arnab Chakravarti, Dietmar Krex, Gabriele Schackert.   

Abstract

PURPOSE: Pseudoprogression (PP) during adjuvant treatment of glioblastoma (GBM) is frequent and is a clinically and radiologically challenging problem. While there are several reports of the frequency of PP in GBM cohorts including mainly patients with primary GBM, there are few data on the incidence of PP in patients with secondary glioblastomas (sGBM). Therefore, the goal of this study was to evaluate the frequency of PP in sGBM. METHODS AND MATERIALS: We retrospectively evaluated the incidence of PP in adult patients with sGBM treated with chemoradiation therapy (CRTx) using temozolomide (TMZ) and sought to assess if there was an association between PP and MGMT promoter methylation status, IDH mutations status, or 1p/19q codeletion. The definition of PP according to the Response Assessment in Neuro-Oncology Working Group was used.
RESULTS: None of the evaluable 15 sGBM patients in our series demonstrated a PP. Of the 9 sGBM patients who received concomitant CRTx with TMZ, 6 patients had the methylated MGMT promoter, and 6 patients had IDH mutations. There also was no PP identified in sGBM patients who received sequential CRTx, irrespective of MGMT or IDH status. The median time of follow-up was 3.4 years after diagnosis of an sGBM, and the median overall survival was 18.2 months (range, 14.3-45.2 months). Three of 15 patients had previously received radiation therapy for their World Health Organization low-grade 2 glioma, while none of them had received chemotherapy at that stage.
CONCLUSIONS: Based on this small series of sGBM patients treated with CRTx (concomitantly or sequentially) the frequency of PP appears to be very low in sGBM, even in those patients with methylated MGMT promoter or IDH mutations. Our results highlight the differences between primary glioblastomas and sGBM in particular as they relate to PP.
Copyright © 2013 Elsevier Inc. All rights reserved.

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Year:  2013        PMID: 24267971      PMCID: PMC4349215          DOI: 10.1016/j.ijrobp.2013.09.036

Source DB:  PubMed          Journal:  Int J Radiat Oncol Biol Phys        ISSN: 0360-3016            Impact factor:   7.038


  18 in total

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Authors:  M C Y de Wit; H G de Bruin; W Eijkenboom; P A E Sillevis Smitt; M J van den Bent
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Review 4.  Clinical features, mechanisms, and management of pseudoprogression in malignant gliomas.

Authors:  Dieta Brandsma; Lukas Stalpers; Walter Taal; Peter Sminia; Martin J van den Bent
Journal:  Lancet Oncol       Date:  2008-05       Impact factor: 41.316

Review 5.  The definition of primary and secondary glioblastoma.

Authors:  Hiroko Ohgaki; Paul Kleihues
Journal:  Clin Cancer Res       Date:  2012-12-03       Impact factor: 12.531

6.  MGMT promoter methylation status can predict the incidence and outcome of pseudoprogression after concomitant radiochemotherapy in newly diagnosed glioblastoma patients.

Authors:  Alba A Brandes; Enrico Franceschi; Alicia Tosoni; Valeria Blatt; Annalisa Pession; Giovanni Tallini; Roberta Bertorelle; Stefania Bartolini; Fabio Calbucci; Alvaro Andreoli; Giampiero Frezza; Marco Leonardi; Federica Spagnolli; Mario Ermani
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7.  Incidence of early pseudo-progression in a cohort of malignant glioma patients treated with chemoirradiation with temozolomide.

Authors:  Walter Taal; Dieta Brandsma; Hein G de Bruin; Jacoline E Bromberg; Annemarie T Swaak-Kragten; Peter A E Sillevis Smitt; Corine A van Es; Martin J van den Bent
Journal:  Cancer       Date:  2008-07-15       Impact factor: 6.860

8.  Type and frequency of IDH1 and IDH2 mutations are related to astrocytic and oligodendroglial differentiation and age: a study of 1,010 diffuse gliomas.

Authors:  Christian Hartmann; Jochen Meyer; Jörg Balss; David Capper; Wolf Mueller; Arne Christians; Jörg Felsberg; Marietta Wolter; Christian Mawrin; Wolfgang Wick; Michael Weller; Christel Herold-Mende; Andreas Unterberg; Judith W M Jeuken; Peter Wesseling; Guido Reifenberger; Andreas von Deimling
Journal:  Acta Neuropathol       Date:  2009-06-25       Impact factor: 17.088

9.  Accumulation of 2-hydroxyglutarate is not a biomarker for malignant progression in IDH-mutated low-grade gliomas.

Authors:  Tareq A Juratli; Mirko Peitzsch; Kathrin Geiger; Gabriele Schackert; Graeme Eisenhofer; Dietmar Krex
Journal:  Neuro Oncol       Date:  2013-02-14       Impact factor: 13.029

10.  The prognostic value of IDH mutations and MGMT promoter status in secondary high-grade gliomas.

Authors:  T A Juratli; M Kirsch; K Geiger; B Klink; E Leipnitz; T Pinzer; S Soucek; E Schrock; E Schrok; G Schackert; D Krex
Journal:  J Neurooncol       Date:  2012-09-27       Impact factor: 4.506

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  3 in total

1.  Radio-chemotherapy improves survival in IDH-mutant, 1p/19q non-codeleted secondary high-grade astrocytoma patients.

Authors:  Tareq A Juratli; Tim Lautenschläger; Kathrin D Geiger; Thomas Pinzer; Mechthild Krause; Gabriele Schackert; Dietmar Krex
Journal:  J Neurooncol       Date:  2015-06-02       Impact factor: 4.130

2.  Characterization of pseudoprogression in patients with glioblastoma: is histology the gold standard?

Authors:  Isaac Melguizo-Gavilanes; Janet M Bruner; Nandita Guha-Thakurta; Kenneth R Hess; Vinay K Puduvalli
Journal:  J Neurooncol       Date:  2015-04-18       Impact factor: 4.130

3.  Isocitrate dehydrogenase 1 mutant glioblastomas demonstrate a decreased rate of pseudoprogression: a multi-institutional experience.

Authors:  Homan Mohammadi; Kevin Shiue; G Daniel Grass; Vivek Verma; Kay Engellandt; Dirk Daubner; Gabriele Schackert; Mercia J Gondim; Dibson Gondim; Alexander O Vortmeyer; Aaron P Kamer; William Jin; Timothy J Robinson; Gordon Watson; Hsiang-Hsuan M Yu; Tim Lautenschlaeger
Journal:  Neurooncol Pract       Date:  2019-10-10
  3 in total

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