Literature DB >> 24057287

Comparison of two mitotane starting dose regimens in patients with advanced adrenocortical carcinoma.

T M Kerkhofs1, E Baudin, M Terzolo, B Allolio, R Chadarevian, H H Mueller, B Skogseid, S Leboulleux, F Mantero, H R Haak, M Fassnacht.   

Abstract

CONTEXT: Mitotane is the only approved drug for treatment of adrenocortical carcinoma. Its pharmacokinetic properties are not fully elucidated and different dosing regimens have never been compared head to head.
OBJECTIVE: The objective of the study was to investigate the relationship between mitotane dose and plasma concentration comparing two dosing regimens. DESIGN/
SETTING: This was a prospective, open-label, multicenter trial of a predefined duration of 12 weeks. PATIENTS/
INTERVENTIONS: Forty mitotane-naïve patients with metastatic adrenocortical carcinoma were assigned to a predefined low- or high-dose regimen by the local investigator. Thirty-two patients could be evaluated in detail. MAIN OUTCOME MEASURE: The difference in median mitotane plasma levels between both treatment groups was measured.
RESULTS: Despite a difference in mean cumulative dose (440 ± 142 g vs 272 ± 121 g), median maximum plasma levels were not significantly different between the two groups [high dose 14.3 mg/L (range 6.3-29.7, n = 20) vs 11.3 mg/L (range 5.5-20.0, n = 12), P = .235]. Ten of 20 patients on the high-dose regimen reached plasma concentrations of 14 mg/L or greater after 46 days (range 18-81 d) compared with 4 of 12 patients on the low-dose regimen after 55 days (range 46-74 d, P = .286). All patients who reached 14 mg/L at 12 weeks displayed a level of 4.1 mg/L or greater on day 33 (100% sensitivity). There were no significant differences in frequency and severity of adverse events. Among patients not receiving concomitant chemotherapy mitotane exposure was higher in the high-dose group: 1013 ± 494 mg/L · d vs 555 ± 168 mg/L · d (P = .080).
CONCLUSIONS: The high-dose starting regimen resulted in neither significantly different mitotane levels nor a different rate of adverse events, but concomitant chemotherapy influenced these results. Thus, for mitotane monotherapy the high-dose approach is favorable, whereas for combination therapy a lower dose seems reasonable.

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Year:  2013        PMID: 24057287     DOI: 10.1210/jc.2013-2281

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  23 in total

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6.  Association of Nrf2 expression and mutation with Weiss and Helsinki scores in adrenocortical carcinoma.

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7.  Time Until Partial Response in Metastatic Adrenocortical Carcinoma Long-Term Survivors.

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8.  Inhibition of Aurora kinase A activity enhances the antitumor response of beta-catenin blockade in human adrenocortical cancer cells.

Authors:  Andrea Gutierrez Maria; Kleiton Silva Borges; R C P Lira; Carolina Hassib Thomé; Annabel Berthon; Ludivine Drougat; Katja Kiseljak-Vassiliades; Margaret E Wierman; Fabio R Faucz; Vitor Marcel Faça; Luiz Gonzaga Tone; Constantine A Stratakis
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9.  Survival and prognostic factors for adrenocortical carcinoma: a single institution experience.

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Review 10.  Adrenocortical carcinoma (ACC): diagnosis, prognosis, and treatment.

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