Literature DB >> 24040897

The lipid peroxidation end-product and oxidant 4-hydroxynonenal induces insulin resistance in rat slow-twitch skeletal muscle.

Mujalin Prasannarong1, Fernando R Santos, Payam Hooshmand, Paria Hooshmand, Franchesca J Giovannini, Erik J Henriksen.   

Abstract

CONTEXT: The lipid peroxidation end-product and oxidant 4-hydroxynonenal (4-HNE) impairs cell function. However, the impact of 4-HNE on the glucose transport system in mammalian slow-twitch skeletal muscle is not known.
OBJECTIVE: We assessed the effects of 4-HNE on insulin signalling and glucose transport activity in slow-twitch muscle by incubating soleus strips from lean Zucker rats with 4-HNE (50 µM) in the absence or presence of insulin (5 mU/ml) for up to 6 hr.
RESULTS: Insulin-stimulated glucose transport activity was significantly (p<0.05) decreased by 4-HNE at 2 hr. AS160 Thr(642) phosphorylation was decreased at 2 hr, whereas Akt Ser473 phosphorylation and IRS-1 protein expression were not substantially changed until 4 hr. IRS-2 protein expression was slightly decreased only at 6 hr.
CONCLUSIONS: The lipid peroxidation end-product and oxidant 4-HNE induces insulin resistance of glucose transport activity in rat slow-twitch skeletal muscle, initially associated with impaired phosphorylation of AS160.

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Year:  2013        PMID: 24040897     DOI: 10.3109/13813455.2013.834937

Source DB:  PubMed          Journal:  Arch Physiol Biochem        ISSN: 1381-3455            Impact factor:   4.076


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