Literature DB >> 23913553

Exon sequencing of PAX3 and T (brachyury) in cases with spina bifida.

A J Agopian1, Angela D Bhalla, Eric Boerwinkle, Richard H Finnell, Megan L Grove, James E Hixson, Lawrence C Shimmin, Anshuman Sewda, Colin Stuart, Yu Zhong, Huiping Zhu, Laura E Mitchell.   

Abstract

BACKGROUND: Based on studies in animals and humans, PAX3 and T (brachyury) are candidate genes for spina bifida. However, neither gene has been definitively identified as a risk factor for this condition.
METHODS: Sanger sequencing was used to identify variants in all PAX3 and T exons and promoter regions in 114 spina bifida cases. For known variants, allele frequencies in cases were compared with those from public databases using unadjusted odds ratios. Novel variants were genotyped in parents and assessed for predicted functional impact.
RESULTS: We identified common variants in PAX3 (n = 2) and T (n = 3) for which the allele frequencies in cases were significantly different from those reported in at least one public database. We also identified novel variants in both PAX3 (n = 11) and T (n = 1) in spina bifida cases. Several of the novel PAX3 variants are predicted to be highly conserved and/or impact gene function or expression.
CONCLUSION: These studies provide some evidence that common variants of PAX3 and T are associated with spina bifida. Rare and novel variants in these genes were also identified in affected individuals. However, additional studies will be required to determine whether these variants influence the risk of spina bifida.
Copyright © 2013 Wiley Periodicals, Inc.

Entities:  

Keywords:  PAX3; T locus; genetic epidemiology; myelomeningocele; sequencing; spina bifida; word

Mesh:

Substances:

Year:  2013        PMID: 23913553      PMCID: PMC3877942          DOI: 10.1002/bdra.23163

Source DB:  PubMed          Journal:  Birth Defects Res A Clin Mol Teratol        ISSN: 1542-0752


  20 in total

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